The Remedy Radio Show Notes with Tony Pantalleresco – To Air 24/02/2012

Another great show expected this week with Tony.  Topics Covered are below:

  • Monsanto guilty of chemical poisoning inFrance
  • Free speech become too expensive!
  • Systematic review- the diagnosis and staging of non-alcoholic fatty liver disease and non-alcoholic steatohepatitis
  • EFSA sets recommended EU protein intakes at 0.8g/kg per dayHome Made Protein Shakes Continue reading
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Weekly Health News Round Up – 10/02/12 (Posted late)

This  is the first blog for weekly round ups in the health and nutrition world. I will be bringing you  the best of the news headlines from the week as well as my top picks of other blogs and websites putting out useful information, as well as media such as youtube videos and pod casts.

so for the first week here goes :

Firstly I want to jump into something that everyone should know about and be aware of and that is GMO foods. Even if you’re not worried about the crap you put into your mouth, you really need to get a little educated on what some companies are trying to feed you and your children.  Here is a short youtube to fille you in on just what Genetically Modified Foods (GMO) are and how they are created :

It is unbelievable and when I tell you that almost all Soy and Corn products are GMO these days you know you must avoid these foods. The damage that these foods have been shown to cause is scary, the fact that we have no idea what the long-term effect will be should get you angry that these foods are being fed to us!

Here is a link to a trailer on you tube for a new film

Now If you want to keep up to day with the latest in health politics, or find out the REAL news then take a look on facebook at Dr Peter Rouse who has a low tolerance thresh hold for government bulls**t  and he take a dislike to large financial corporations that make money from making us sick!!  Check out his Facebook here!/profile.php?id=1708172500

Below are some interesting news stories from this week for you to check out.

Science Daily – Drug Quickly Reverses Alzheimer’s Symptoms in Mice

Natural News – FDA monopoly enforcement goes after Google for $500 million in online pharmacy ad profits

And for this week, as things have got a bit hectic that will be it.   This will be getting more regular here, with lots of new stories, information and hopefully some cheap deals on quality supplements.

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Things To Come…

Hello All (I’m assuming at least three people might stumble across this page which means ALL)

This Blog is going to get started in a big way in 2012.  I will be updating it until then with information from Tony Pantalleresco’s web site to give you some good information to be getting on with.

I should introduce myself. My name is John Stephens and I have been interested in nutrition for around 10 years now, almost three years in a serious way. I found Tony Pantalleresco on YouTube and watched all of his video clips. I contacted Tony and have been talking and learning from him ever since.  Tony has been in the health industry for 30+ years and keeps surprising me with his depth of knowledge and he always seems to be ahead of the curve. The reason for this is because he has no agenda. He isn’t a paleo enthusiast (nothing wrong with that i love the paleo concept) and he is not a juicing fanatic, or a vegan or part of any other group that can be labeled, he just looks for the truth and he looks at the current research as well as some historical research (when research was carried out to find answers and not to find the answer that the pharmaceutical company sponsor would like to be found) and with that he gets tends to find the answers. No agenda, just truth.

Apart from the amazing knowledge Tony P has the one thing I admire from Tony is his pure work ethic and  morals. Tony has a Shop in his home town in Canada but he gives away  TONS of free information on You Tube (Search herbsplusbeadswork) and on his radio show on the  Micro Effect. (his show is called The Remedy)  Tony gives advice on how to use every day items to augment your health without expensive supplements.

I am still very much a student in my eyes but I have been around enough to have some great contacts and places for information on Health, nutrition and Longevity. I will bring you the best links and information from other blogs, news sites etc.

in 2012 Tony will be writing articles, posting his radio show notes, Giving you remedies Answering questions and telling you all the latest political news with regards to your health and your rights with regards to access to safe foods, water and supplements. You only have to look whats going on in the US with the demonization of raw milk to see that some thing is definitely wrong.

Any way, Lots to come so pop back and if you have any questions, please feel free to ask!

Until Then I will be moving information from

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Getting Back To It, and Site Migration!

Ok, So I am working to make this blog a priority for me to keep it updated and to get it to where is was always supposed to be, posting food for thought, but more importantly, helping people sift through the confusion world of nutrition and health! With  papers printing that this will give you cancer and that will cure cancer,  and then one radio show promoting veganism and another promoting 80% meat diet, no one knows what the hell to do any more. And My self and Mr Tony Pantalleresco want to help. Without bias.

Any how, thats not been the case. We have both been busy and this blog got loaded with a lot of Mr P’s info from his site and then not much more.. That is changing NOW!

Firstly, My site,  was hacked early this year and has been having problems ever since. Right now it looks like i’m in for a complete new install, which i am not happy about, but such is life. So for now, I will be posting interesting and Health news articles here weekly from various sites.  I read and follow many sites and there are a lot of good storied out there, but also a lot of rubbish. I’ll sift through and find you the good stuff. You still might get a little contradiction, but then health is not black and white. I wont be posting propaganda articles!

Tony Has been doing some great radio shows recently with some superb info so ill get the show notes up here very soon.

Thanks for checking in.

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Holiday Time

Ok, so this blog has been ticking over with some of Mr Pantalleresco’s  Radio show notes. Not much in the way of regular blogging as there has been soo much going on. BUT …


On the 1st I leave the UK to head off to sunny Las Vegas to get married. I’ m away for three weeks but once I am back , my studies are done so i will be igniting this blog! After a year or so after starting this blog up, MR P have finally found time to sign up and will begin posting here regularly!  I will try to organise  the crazy ramblings of the genius of  the man known as The Remedy, but don’t hold me to that!


Ok , so, i’ll be back in about a month, Tony might pop up in the mean time.


Wish me luck…

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Tony Pantalleresco Show of the Month October 19-2012



Coffee Drinkers Have Lower Risk of Death- Study Suggests

Potato Storage- Essential Oils as Antigerminants

Top 10 Foods Highest in Potassium

EFSA publishes initial review on GM maize and herbicide study

Claims on many supplements don’t comply with law, report says


Coffee Drinkers Have Lower Risk of Death- Study Suggests

A new study found that older adults who drank coffee — caffeinated or decaffeinated — had a lower risk of death overall than others who did not drink coffee.

ScienceDaily (May 19, 2012) — Older adults who drank coffee — caffeinated or decaffeinated — had a lower risk of death overall than others who did not drink coffee, according a study by researchers from the National Cancer Institute (NCI), part of the National Institutes of Health, and AARP.—Coffee drinkers were less likely to die from heart disease, respiratory disease, stroke, injuries and accidents, diabetes, and infections, although the association was not seen for cancer. These results from a large study of older adults were observed after adjustment for the effects of other risk factors on mortality, such as smoking and alcohol consumption. Researchers caution, however, that they can’t be sure whether these associations mean that drinking coffee actually makes people live longer. The results of the study were published in the May 17, 2012 edition of the New England Journal of Medicine.—Neal Freedman, Ph.D., Division of Cancer Epidemiology and Genetics, NCI, and his colleagues examined the association between coffee drinking and risk of death in 400,000 U.S. men and women ages 50 to 71 who participated in the NIH-AARP Diet and Health Study. Information about coffee intake was collected once by questionnaire at study entry in 1995-1996. The participants were followed until the date they died or Dec. 31, 2008, whichever came first.—The researchers found that the association between coffee and reduction in risk of death increased with the amount of coffee consumed. Relative to men and women who did not drink coffee, those who consumed three or more cups of coffee per day had approximately a 10 percent lower risk of death. Coffee drinking was not associated with cancer mortality among women, but there was a slight and only marginally statistically significant association of heavier coffee intake with increased risk of cancer death among men.–“Coffee is one of the most widely consumed beverages in America, but the association between coffee consumption and risk of death has been unclear. We found coffee consumption to be associated with lower risk of death overall, and of death from a number of different causes,” said Freedman. “Although we cannot infer a causal relationship between coffee drinking and lower risk of death, we believe these results do provide some reassurance that coffee drinking does not adversely affect health.”—The investigators caution that coffee intake was assessed by self-report at a single time point and therefore might not reflect long-term patterns of intake. Also, information was not available on how the coffee was prepared (espresso, boiled, filtered, etc.); the researchers consider it possible that preparation methods may affect the levels of any protective components in coffee.—“The mechanism by which coffee protects against risk of death — if indeed the finding reflects a causal relationship — is not clear, because coffee contains more than 1,000 compounds that might potentially affect health,” said Freedman. “The most studied compound is caffeine, although our findings were similar in those who reported the majority of their coffee intake to be caffeinated or decaffeinated.”—Story Source-The above story is reprinted from materials provided byNational Institutes of Health. —Journal Reference-Neal D. Freedman, Yikyung Park, Christian C. Abnet, Albert R. Hollenbeck, Rashmi Sinha. Association of Coffee Drinking with Total and Cause-Specific Mortality. New England Journal of Medicine, 2012; 366 (20): 1891 DOI: 10.1056/NEJMoa1112010


Potato Storage- Essential Oils as Antigerminants

ScienceDaily (Oct. 5, 2012) — One of the critical moments in the final quality of the potato occurs during its storage, as there exists the risk of sprouting or rotting due to pathogenic agents such as bacteria and fungi. In order to avoid this, agricultural engineer David Gómez Castillo carried out research for his PhD on the possibility of substituting the current use of chemical products by treating the tuber with essential oils of mint, caraway, coriander, eucalyptus and clove, “which have proved to be great potential inhibitors in the main problems detected.”—The chemical product Clorprofam (CIPC) is the most commonly used as a sprout suppressant on stored potatoes. Nevertheless, possible reductions in permitted dosages, market and consumer pressures seeking healthier and, moreover, more environmentally-friendly products, have made it necessary to find alternatives to these synthetic products, with the market, culinary and technological quality of the potato remaining unaltered.—This is the context of the research by David Gómez Castillo, who has evaluated alternative treatment using essential oils of mint, caraway, coriander, eucalyptus and clove. In concrete, he studied the effect of applying these oils with table-stock varieties of the potato (Agata and Monalisa) and industrial ones (Agria and Kennebec), and compared the results thereof with those that had been treated chemically.

A good alternative—The research analysed two parameters: the commercial quality (germination, texture and colour of the tuber) and the culinary and technological quality (colour and texture of slices of the potato, dry material, total soluble solids, reductor sugars and sensorial analysis). Evaluations at 10, 25, 40, 55 and 70 days in storage were also undertaken, the antimicrobial effect of essential oils being assessed for the principal phytopathogens (fungi and bacteria).—According to Mr Gómez, “we found a high antigerminant capacity with treatment using the essential oil of coriander for industrial crops, and with the essential oil of mint for both industrial and table-stock cropsThese showed great inhibitory potential on the principal phytopathogenic problems studied and all this makes a good alternative to CPIC use for storage of potatoes.”—It was also shown that the essential oil of eucalyptus, for its high antigerminant capacity with table-stock potatoes, “could be another alternative for reducing post-harvest losses due to phytopathogenic problems, obtaining even better results if the treatment is accompanied by the essential oil of clove.”–In the opinion of this researcher, the use of treatment with essential oils in the storage of potatoes “can provide added value in the application of antigerminant treatment, due to its efficacy in controlling the progress of important phytopathogens.”—Story Source-The above story is reprinted from materials provided by Basque Research.

Recipe for AntiPhytopathegenic for Potato’s—take either a combination of peppermint  and coriander add 2 drops each to either a Sprayer with water—make sure the Essential oils are dispersed well in the water—blend in a blender for about 2 minutes to mix—once done then spray the potatoes being stored—or take a vaporiser and add the oils into the vaporiser  and allow it to mist the air as well—this will cause the components to be air borne and will reduce the break down—-this same principle can be done with other things as well or even utilize this principle to do a room or household to eliminate pathogens that might be airborne—keep the potatoes in a cool place for storage as well will reduce spoilage


Top 10 Foods Highest in Potassium

Potassium is an essential nutrient used to maintain fluid and electrolyte balance in the body. A deficiency in potassium causes fatigue, irritability, and hypertension (increased blood pressure). Overdose of potassium from natural sources is nearly impossible, however, it is possible to consume too much potassium via potassium salts which can lead to nausea, vomiting, and even heart attack. Potassium from natural food sources, like the ones listed below, are considered safe and healthy. The current percent daily value for potassium is a whopping 3.5 grams, below is a list of high potassium foods. For more foods high in potassium please see the lists of potassium rich foodsfruits high in potassium, and vegetables high in potassium.

#1: Dried Herbs
Long used for medicinal purposes, herbs are packed with nutrients and potassium is no exception. Dried Chervil contains the most potassium with 4.7g (135% DV) per 100g serving, or 95mg (3% DV) per tablespoon. It is followed by Dried Coriander (3% DV) per tblsp, Dried Parsley (2% DV), Dried Basil, Dried Dill, Dried Tarragon, Ground Turmeric, Saffron, and finally Dried Oregano with 50mg (1% DV).
Click to see complete nutrition facts

#2: Avocados
Avocados are great when made into guacamole or in a salad. 100 grams will provide 485mg of potassium or 14% of the DV. That is 1.1g (32% DV) in one cup pureed, and 975mg (28% DV) in a single avocado (201 grams).
Click to see complete nutrition facts || More Fruits High in Potassium

#3: Paprika and Red Chili Powder
Either paprika or red chili powder add a nice kick to any dish, and with all the potassium they provide you have good reason to start adding them. Paprika provides the most potassium with 2.3g (67% DV) per 100 gram serving, or 164mg (5% DV) per tablespoon. Chili powder will provide 1.9g (55% DV) per 100 gram serving or 153mg (4% DV) per tablespoon. Click to see complete nutrition facts

#4: Cocoa Powder and Chocolate
Dark chocolate is an excellent source of iron and zinc in addition to potassium. Pure cocoa powder without any fat, milk, or sugar, provides the most potassium with 1.5 grams (44% DV) in a 100g serving, or 1.3g (37% DV) per cup, and 76mg (2% DV) per tablespoon. Unsweetened baking chocolate provides 830mg (24% DV) per 100 gram serving or 241mg (7% DV) per square. Most sweetened milk chocolates will provide around 272mg (11% DV) per 100 gram serving, and 164mg (5% DV) per bar (1.5oz). Click to see complete nutrition facts

#5: Dried Apricots, Prunes, Zante Currants, and Raisins
Most common as a snack, dried apricots and prunes can also be chopped and served in a salad. A good source of fiber and many other vitamins, apricots provide 1.9g (53%DV) of potassium per 100g serving (about 20 dried apricots). Prunes provide 1g (30% DV) per 100g serving, or 1.4g (40% DV) per cup. Zante currants are really a type of grape and taste very similar to raisins. Zante currants provide 892mg (25% DV) of potassium per 100g serving, or 1.3g (37% DV) per cup. Raisins provide almost the same amount with 825mg (24% DV) per 100 gram serving, or 1.2g (24% DV) per cup. Click to see complete nutrition facts

#6: Pistachios and Other Nuts
Pistachios are a delicious snack, and a great addition to salads. 100 grams (~3/4cup) will provide 1g (30% DV) of potassium. Other nuts high in potassium include Beechnuts (29% DV per 100g), Ginko nuts (29% DV), Chestnuts (28% DV), Almonds (21% DV), Hazelnuts (19% DV), Cashews (18% DV), Pine nuts (17% DV), Coconuts (16% DV), and Walnuts (15% DV).
Click to see complete nutrition facts

#7: Seeds (Pumpkin, Squash, Sunflower, and Flax)
A popular food in the Middle East and East Asia pumpkin and squash seeds contain about 919mg (26% DV) of potassium per 100g serving, 588mg (17% DV) per cup. If you can’t find these in your local supermarket you will surely find them in Middle Eastern or East Asian specialty stores. Alternatively, you can also save any pumpkin and squash seeds you have and roast them in your oven. The seeds are typically eaten by cracking the outer shell and eating the seed inside. Sunflower seeds are also a good source of potassium, providing 850mg (24% DV) per 100 gram serving, or 1.1g (31% DV) per cup. Flax seeds provide 813mg (23% DV) of potassium per 100 gram serving, or 1.4g (39% DV) per cup, and 81mg (2% DV) per tablespoon.
Click to see complete nutrition facts. Buy Pumpkin Seeds from

#8: Fish (Pompano, Salmon, Halibut, Tuna)
Fish has many health benefits and is a great source of potassium. Pompano provides the most with 636mg (18% DV) per 100 gram serving, or 540mg (15% DV) per fillet (3 ounces, 85 grams). It is followed by Salmon which provides 534mg (15% DV) per 3 ounce serving, Halibut, Yellow Fin Tuna, Lingcod, Mackerel, Anchovies, Herring, Cod, Snapper, Rockfish, Tilefish, Grouper, and finally Trout with 394mg (11% DV) in a 3 ounce serinvg. Cooking fish with dry heat is the best way to preseve the potassium content. Click to see complete nutrition facts

#9: Beans
White beans provide the most potassium with 561mg (16% DV) per 100 gram serving, 1g (29% DV) per cup cooked. White beans are followed by Adzuki Beans, Soy Beans, Lima Beans, Pinto Beans, Kidney Beans, Great Northern Beans, Navy Beans, Pigeon Peas, Cranberry (Roman) Beans, French Beans, Lentils, Split Peas, Black Beans, Hyancinth, and finally Yardlong Beans with 539mg (15% DV) per cup cooked.
Click to see complete nutrition facts

#10: Dates (Medjool)
Dates are great as a snack, as an addition to fruits salads, or even savory stews. Medjool dates provide 696mg (20% DV) per 100 gram serving, or 167mg (5% DV) in a single date.
Click to see complete nutrition facts



EFSA publishes initial review on GM maize and herbicide study

The European Food Safety Authority has concluded that a recent paper raising concerns about the potential toxicity of genetically modified (GM) maize NK603 and of a herbicide containing glyphosate is of insufficient scientific quality to be considered as valid for risk assessment.—EFSA’s initial review found that the design, reporting and analysis of the study, as outlined in the paper, are inadequate. To enable the fullest understanding of the study the Authority has invited authors Séraliniet al to share key additional information.–Such shortcomings mean that EFSA is presently unable to regard the authors’ conclusions as scientifically sound. The numerous issues relating to the design and methodology of the study as described in the paper mean that no conclusions can be made about the occurrence of tumours in the rats tested.—Therefore, based on the information published by the authors, EFSA does not see a need to re-examine its previous safety evaluation of maize NK603 nor to consider these findings in the ongoing assessment of glyphosate.—EFSA assessed the paper against recognised good scientific practices, such as internationally agreed study and reporting guidelines.—-Per Bergman, who led EFSA’s work, said: “Some may be surprised that EFSA’s statement focuses on the methodology of this study rather than its outcomes;however, this goes to the very heart of the matter. When conducting a study it is crucial to ensure a proper framework is in place. Having clear objectives and the correct design and methodology create a solid base from which accurate data and valid conclusions can follow. Without these elements a study is unlikely to be reliable and valid.”—The Director of Scientific Evaluation of Regulated Products added that the consideration of possible long-term effects of GMOs has been, and will continue to be, a key focus of EFSA’s work to protect animals, humans and the environment.—-EFSA’s preliminary review issued today is the first step in a two-stage process. A second analysis will be delivered by the end of October 2012.  This will take into account any additional information from the study authors, who will be given an opportunity to supply study documentation and procedures to the Authority to ensure the broadest possible understanding of their work. It will also include an overview of Member State assessments of the paper and an analysis from the German authorities responsible for the assessment of glyphosate.

Main findings of Initial Review—The task force, whose members were drawn from the Authority’s GMO, pesticide and scientific assessment units, has outlined a list of issues about the paper that would need to be resolved before it could be viewed as well-conducted and properly-reported study.

  • The strain of rat used in the two-year study is prone to developing tumours during their life expectancy of approximately two years. This means the observed frequency of tumours is influenced by the natural incidence of tumours typical of this strain, regardless of any treatment. This is neither taken into account nor discussed by the authors.
  • The authors split the rats into 10 treatment sets but established only one control group. This meant there was no appropriate control for four sets – some 40% of the animals – all of whom were fed GM maize treated or not treated with a herbicide containing glyphosate.
  • The paper has not complied with internationally-recognised standard methods – known as protocols – for setting up and carrying out experiments. Many of these procedures are developed by the OECD (Organisation for Economic Cooperation and Development).
  • For a study of this type, the relevant OECD guideline specifies the need for a minimum of 50 rats per treatment group. Séralini et al used only 10 rodents per treatment set. The low number of animals used is insufficient to distinguish between the incidence of tumours due to chance rather than specific treatment effects.
  • The authors have not stated any objectives, which are the questions a study is designed to answer. Research objectives define crucial factors such as the study design, correct sample size, and the statistical methods used to analyse data – all of which have a direct impact on the reliability of findings.
  • No information is given about the composition of the food given to the rats, how it was stored or details of harmful substances – such as mycotoxins – that it might have contained.
  • It is not possible to properly evaluate the exposure of the rats to the herbicide as intake is not clearly reported. The authors report only the application rate of the herbicide used to spray the plants and the concentration added to the rats’ drinking water but report no details about the volume of the feed or water consumed.
  • The paper does not employ a commonly-used statistical analysis method nor does it state if the method was specified prior to starting the study. The validity of the method used is queried and there are questions over the reporting of tumour incidence. Important data, such as a summary of drop outs and an estimation of unbiased treatment effects have not been included in the paper.
  • Many endpoints – what is measured in the study – have not been reported in the paper. This includes relevant information on lesions, other than tumours, that were observed. EFSA has called on the authors to report all endpoints in the name of openness and transparency.

Notes to editors:

EFSA set up a multi-disciplinary task force in response to an urgent request from the European Commission to evaluate a paper by Séralini et al to assess whether its findings could lead the Authority to reconsider its previous opinion on maize NK603. The two-year study, published in the journal Food and Chemical Toxicology on 19 September 2012, has suggested that consumption of the GM maize and a herbicide containing glyphosate at levels below officially-safe limits are linked to a reported increase in incidence of tumours in rats.


Claims on many supplements don't comply with law, report says
The structure function claims on many dietary supplement products do not comply with federal law,
 a government report released on WednesdayThe[U1]  report recommends greater regulatory 
powers for FDA to bring products into compliance[U2] .---Industry sources reacted negatively to the assertion
 that FDA needs new statutory powers, but were more accommodating on the report’s suggestions on how 
the claims notification process should be tightened up[U3] . The report, conducted by the Office of Inspector 
General of the United States Department of Health and Human Services, looked at the claims on 127 dietary s
upplements in the weight loss and immune support The report looked at the label language and what kind 
of evidence was cited to back up the claims, to judge compliance with FDA-mandated notifications and disclaimers 
and to see how many claims trended over into prohibited disease claim territory. The report said that the
 popularity of structure function claims is on the rise, and maintains that problems with the use 
of these claims is on the rise, too. Report cites thin supporting evidence The report’s broad conclusions 
were these: Many claims were not backed by evidence from human studies[U4] . Of the human studies 
supplied by manufacturers in response to HHS requests, few appeared to satisfy FDA recommendations 
in every respect in terms of study design and relevance to the meaning of the claim[U5] . It also found 
that 20% of the supplements in the sample were making prohibited disease claims and that 7% lacked 
the disclaimer that is supposed to accompany every structure function claim. The report went on to 
recommend that FDA should seek additional statutory authority to regulate label claims to make sure 
that suitable evidence exists to back up the claim, to make sure that proper label notifications are
 in place and to make sure companies are not making illegal disease claims In[U6]  addition, it said FDA
 needs to clean up its tracking of who is making what claims, and who has complied with the requirement of a 30-day
 prior notification of the use of a claim on a product. Immediate reaction  Report overreaches Reaction from trade
 groups and industry observers was swift and decisive. The Office of Inspector General reviewed just 127 supplements 
out of an estimated 29,000 products on the market. This small sample of supplements shouldn’t smear the entire industry, 
said John Shaw, executive director and CEO of the Natural Products Foundation. The president and CEO of the
 organization, said, What’s most disappointing is that this was not a random sampling [of the industry].The vast majority
 of the industry, including our members, is doing the right thing. These reports give good companies a black eye, 
he said.The small sample size and big conclusions also was an issue for Marc Ullman, who has worked with the 
" Natural Products Foundation’s Truth in Advertising  industry self-regulation effort. They are recommended sweeping 
changes to the law, the imposition of vast new regulatory burdens on FDA based on the fact that (a small number)
 of dietary supplements didn’t pass substantiation. To me it seems quite a reach, Ullman, an attorney with the New
 York-based firm Ullman, Shapiro Ullman, told a reporter. I cannot fathom the kind of broad generalizations they 
made based on this kind of sample size. Nuanced response to report’s recommendations Regarding the recommendations, 
Mister said CRN was very supportive of FDA to do more, especially when it came keeping better 
track of health claim notifications, which companies are supposed to send in 30 days prior to a 
product hitting the  But the first recommendation, calling for FDA to ‘statutory authority to review
 substantiation for structure/function claims to determine whether they are truthful and not 
misleading[U7] ,’ is a non-starter, he said.This sounds like pre-market approval to us, Mister said. I do hope that 
FDA sees that some of this is targeted towards them, he added. The agency needs to manage the information they 
already have. Structure/function claims and registrations need to be catalogued to be accessible.The methodology
 of the report divided up the supplements more or less equally between the weight loss and immune sectors, and
 also about equally between supplements purchased in retail outlets on the Internet. This last detail interested Tony 
Young, legal counsel for the American Herbal Products Association, especially in relation to the finding that 20% of 
products were making disease claims. An interesting question would be whether those were products purchased off the 
Internet or in retail stores. We expect that there is a higher standard out there to get products on retail shelves and
 that most of the major retailers don’t carry products that make that kind of claim, he said. No public health risk At
 the end of the day, Young said, there is no imminent public health risk in the report’s findings. Whether FDA should 
expend substantial resources doing the kinds of things that were suggested is a decision that FDA would have to make
 with regard to all of the other public health priorities,” he said.The report might go overboard in its enforcement
 recommendations, Ullman said, but that doesn’t obviate legitimate questions about how some companies market their 
products As an industry we need to recognize—and we do recognize—that there is a problem, he said, going on to cite NPF’s 
Truth in Advertising effort and CRN’s cooperation with the ""
 National Advertising Division as appropriate ways to self-police label claims.Everybody in our industry argues that there
 should be more enforcement with respect to unlawful disease claims so there is pretty much unanimity on that, 
Young said[U8] .


[U1]A new attack on supplements

 [U2]Drug Companies want to debilitate the industry further for easier acquisition of that health food industry market—where the owners will surrender there companies and sell out

[U3]This is the HFI-health food industry caving in  and surrendering—the industry should be looking after itself without gov’t interference—

 [U4]This is the EFSA—european food and safe authority nonsence—this is the FDA compliance  with the EFSA

[U5]Again EFSA

 [U6]Gov’t interference—again to debilitate the industry to a point where they either bail out or surrender—the health food industry as well a the consumers need to collaborate and get rid of  the common enemy Gov’t

[U7]Is this not the dumbest thing you ever heard? The wolf guarding the hen house—the industry is going to ask the gov’t to monitor the competition??? What a deception here

[U8]This is really disappointing—the big players what the FDA to do there dirty work for them so they can eliminate all competition and have it all under 1 umbrella this is what is really going on a destruction of a free enterprise effect and everyone has to comply  and the independent is going to either be absorbed or eliminated from the market

 [U1]Smart Meter-Cell Phone—HAARP -Microwaves

 [U2] A new attack on supplements

 [U3]Drug Companies want to debilitate the industry further for easier acquisition of that health food industry market—where the owners will surrender there companies and sell out

 [U4]This is the HFI-health food industry caving in  and surrendering—the industry should be looking after itself without gov’t interference—

 [U5]This is the EFSA—european food and safe authority nonsence—this is the FDA compliance  with the EFSA

 [U6]Again EFSA

 [U7]Gov’t interference—again to debilitate the industry to a point where they either bail out or surrender—the health food industry as well a the consumers need to collaborate and get rid of  the common enemy Gov’t

 [U8]Is this not the dumbest thing you ever heard? The wolf guarding the hen house—the industry is going to ask the gov’t to monitor the competition??? What a deception here

 [U9]This is really disappointing—the big players what the FDA to do there dirty work for them so they can eliminate all competition and have it all under 1 umbrella this is what is really going on a destruction of a free enterprise effect and everyone has to comply  and the independent is going to either be absorbed or eliminated from the market

Posted in Health Politics, Nutrition, Remedies, The Remedy Show notes, Tony Pantellresco's Articles | Leave a comment

Tony Pantalleresco Script of the Show October 15-2012

Inactivation of human immunodeficiency virus by Betadine products & chlorhexidine.

The action of three antiseptics/disinfectants against enveloped and non-enveloped viruses


Aloe + Bioflavonoid ( Pectin )

PARSLEY, THE NEW PREDNISONE— Parsley– Antiinflammatory


Inactivation of human immunodeficiency virus by Betadine products & chlorhexidine.

Harbison MA, Hammer SM.

J Acquir Immune Defic Syndr . 1989;2(1):16-20.

Infectious Disease Section, New England Deaconess Hospital, Boston, Massachusetts.

Eleven povidone-iodine-containing products (Betadine) and chlorhexidine gluconate solution were tested for their ability to inactivate human immunodeficiency virus (HIV) in a cell culture system. All Betadine products completely inactivated the virus at povidone-iodine concentrations of greater than or equal to 0.5% (10- to 20-fold dilutions of stock) except for Betadine Lubricating Antiseptic Gel, which required 2.5% for efficacy (1:2 dilution).


The action of three antiseptics/disinfectants against enveloped and non-enveloped viruses.

Wood A, Payne D.

J Hosp Infect . 1998 Apr;38(4):283-95.

Results indicate that all products were effective in inactivating the enveloped viruses herpes simplex virus type 1 and human immunodeficiency virus type 1, whilst being ineffective in inactivating human coronavirusŠ..Four antiseptic/disinfectant solutions with chloroxylenol, benzalkonium chloride, cetrimide/chlorhexidine and povidone-iodine were also assessed for antiviral effect against human immunodeficiency virus in the presence of whole human blood. All four solutions proved to be effective within 1 min despite the cytotoxic nature of the compounds to the detection system. —Note from Mark Konlee: A few years ago I talked with 2 persons from different parts of the country who have been on an unusual diet for several years with striking similarities and results. They ate no meat and ate seafood, fish and or sea vegetables daily for several years. They were also both HIV positive and non-progressors and neither person had used prescribed antiviral drugs for HIV. Could the iodine and other trace minerals in the seafood they consumed have had something to do with their status as non-progressors? Could eating sea vegetables and ocean fish daily help stop HIV



Andrew Goldsworthy July  2011


There is increasing evidence that wireless transmissions have biological effects, some of which are harmful, at levels that may be orders of magnitude below present safety guidelines. These guidelines were drawn up on the assumption that the radiation could only damage living tissues if it generated significant heat. It has since been shown that radiation at much lower levels has direct electrical effects. These are mainly on electrically charged cell membranes, where the low frequency pulses from the modulated microwaves make them vibrate and leak. This can give rise to many “modern illnesses” ranging from electromagnetic hypersensitivity to cancer and disorders of the immune system. The most dramatic increase in the incidence of autism due to damage to the developing brains of the fetus  and young children. Modulated microwaves, such as those from cell phones, portable phones, WiFi,  baby monitors and wireless smart meters are sources of potentially damaging radiation. The strength of the radiation appears to be less important than the duration and pattern of the exposure, with intermittent and repeated exposure being the most damaging. The strong regular transmissions from wireless smart meters are particularly harmful and more likely to lead to DNA damage, cancer and autism.

Sub-thermal effects of electromagnetic radiation.

There are thousands of scientific papers showing biological effects of non-ionizing electromagnetic radiation occurring well below the levels at which them can generate significant heat.  Many of these have been reviewed at by expert scientist at  and . They include harmful effects such as damage to DNA in living cells that can lead to cancer, loss of fertility, brain damage due to the disruption of the blood-brain barrier and neuronal hyperactivity leading to autism in children. Many of these effects can be attributed to the loss of structurally important calcium from cell membranes, which makes them leak. This can disrupt normal metabolism and also release DNase (which destroys DNA) from the internal structures (lysosomes) that normally recycle waste into the rest of the cell  .

Prolonged and intermittent radiation causes more damage

The duration of the radiation seems to be more important than its strength, with the effects being cumulative as more and more cells are damaged. Interestingly, DNA damage from cell phone radiation is greater when the exposure is intermittent (5 minutes on, 10 minutes off) than when continuous (Diem et al 2005). This may be because the cells are constantly adapting and using energy to defend themselves; they drop their guard during the off period and are caught unawares when it goes on again. This constant switching uses more energy, which eventually leaves the cells less able to counteract the effects of the radiation.

Diem et al. (2005) also found that the effect on DNA damage was still greater if the microwaves were pulsed or modulated to carry information (modulation involves sudden stops and starts of the signal, which are even more damaging).

Smart meters, which operate 24/7 and radiate modulated microwaves intermittently, can therefore be expected to be particularly harmful to DNA.

Microwave radiation causes cancer

There is already evidence that heavy cell phone users are more prone to brain cancers. This has resulted in cell phones now being rated by the World Health Organisation as class 2B carcinogens.  This rating may later be increased, since brain tumours normally take decades to develop and few people have been regularly using a cell phone for more than a single decade. Particularly worrying is the finding by Hardell  and Carlberg (2009 ) that young people were about 5-times more likely to get brain cancer both from cordless and cell phones if they began using them before the age of 20.  The regular transmissions from wireless smart meters can be expected to have much the same effect, with younger people being more at risk. This is possibly because their brain structure is still growing and developing and therefore more susceptible to damage leading to cancer.

The effect of microwaves on autism is far worse

The greatest damage from microwaves is when the brain is first developing in the foetus and the very young child, when it can lead to autism. Dr Dietrich Klinghardt has recently shown the relationship between microwaves and autism; a summary of his work can be found at .

 What is autism?

Autism is in fact a group of life-long disorders (autistic spectrum disorders or ASD) caused by brain malfunctions and is associated with subtle changes in brain anatomy (see Amaral et al. 2008 for a review). The core symptoms are an inability to communicate adequately with others and include abnormal social behaviour, poor verbal and non-verbal communication, unusual and restricted interests, and persistent repetitive behaviour. There are also non-core symptoms, such as an increased risk of epileptic seizures, anxiety and mood disorders. ASD has a strong genetic component, occurs predominantly in males and tends to run in families.

Genetic ASD may be caused by calcium entering neurons

It has been hypothesised that some genetic forms of ASD can be accounted for by known mutations in the genes for ion channels that result in an increased background concentration of calcium in neurons. This would be expected to lead to neuronal hyperactivity, the formation of sometimes unnecessary and inappropriate synapses, which in turn can lead to ASD (Krey and Dolmetsch 2007).

Electromagnetic fields let calcium into neurons too

There has been a 60-fold increase in ASD in recent years, which cannot be accounted for by improvements in diagnostic methods and can only be explained by changes in the environment.  This increase corresponds in time to the proliferation of mobile telecommunications, WiFi, and microwave ovens as well as extremely low frequency fields (ELF) from mains wiring and domestic appliances. We can now explain this in terms of electromagnetically-induced membrane leakage leading to brain hyperactivity and abnormal brain development.

Non-ionising radiation makes cell membranes leak

The first effect of non-ionising electromagnetic radiation is to generate small alternating voltages across the cell membranes, which destabilize them and make them leak. This can have all sorts of consequences as unwanted substances diffuse into and out of cells unhindered, and materials in different parts of the cell that are normally kept separate, become mixed.

Why weak fields are more damaging than strong ones

We have known since the work of Suzanne Bawin and her co-workers (Bawin et al. 1975) that modulated radio-frequency electromagnetic radiation that is far too weak to cause significant heating can nevertheless remove calcium ions (positively charged calcium atoms) from cell membranes in the brain. Later, Carl Blackman showed that this also occurs with extremely low frequency electromagnetic radiation (ELF) but only within one or more “amplitude windows”, above and below which there is little or no effect (Blackman et al. 1982; Blackman 1990).  A proposed molecular mechanism for this can be found in Goldsworthy (2010). In particular, it explains why weak electromagnetic fields can have a greater effect than strong ones and why prolonged exposure to weak fields (where cells are maintained in the unstable condition for longer) is potentially more damaging than relatively brief exposure to much stronger ones.

How calcium ions stabilize cell membranes

This loss of calcium is important because calcium ions bind to and stabilize the negatively charged membranes of living cells. They sit between the negatively charged components of the cell membrane and bind them together rather like mortar binds together the bricks in a wall. Loss of just some of these calcium ions destabilize the membrane and make it more inclined to leak, which can have serious metabolic consequences. Among these are the effects of membrane leakage on the neurons of the brain.

How membrane leakage affects neurons

Neurons transmit information between one another in the form of chemical neurotransmitters that pass across the synapses where they make contact. However, the release of these is normally triggered by a brief pulse of calcium entering the cell. If the membrane is leaky due to electromagnetic exposure, it will already have a high internal calcium concentration as calcium leaks in from the much higher concentration outside.  The effect of this is to put the cells into hair-trigger mode so that they are more likely to release neurotransmitters and the brain as a whole may become hyperactive (Beason and Semm 2002; Krey and Dolmetsch 2007, Volkow et al. 2011). This may not be a good thing since the brain may become overloaded leading to a loss of concentration and what we now call attention deficit hyperactive disorder (ADHD).

How does this impact on autism?

Before and just after its birth, a child’s brain is essentially a blank canvas, and it goes through an intense period of learning to become aware of the significance of all of its new sensory inputs, e.g. to recognise its mother’s face, her expressions and eventually other people and their relationship to him/her (Hawley & Gunner 2000). During this process, the neurons in the brain make countless new connections, the patterns of which store what the child has learnt. However, after a matter of months, connections that are rarely used are pruned automatically (Huttenlocher & Dabholkar 1997) so that those that remain are hard-wired into the child’s psyche. The production of too many and often spurious signals due to electromagnetic exposure during this period will generate frequent random connections, which will also not be pruned, even though they may not make sense. It may be significant that autistic children tend to have slightly larger heads, possibly to accommodate unpruned neurons (Hill & Frith 2003).

Because the pruning process in electromagnetically-exposed children may be more random, it could leave the child with a defective hard-wired mind-set for social interactions, which may then contribute to the various autistic spectrum disorders. These children are not necessarily unintelligent; they may even have more brain cells than the rest of us and some may actually be savants. They may just be held back from having a normal life by a deficiency in the dedicated hard-wired neural networks needed for efficient communication.

Autism and the economy

The incidence of autism has increased 60-fold, in parallel with the increase in electromagnetic pollution over the last thirty years. The chance of having an autistic child may now be as high as one in fifty. Apart from the personal tragedies for the affected children and their families, autism is of enormous economic importance. In the UK alone, the annual cost to the Nation in care and lost production exceeds the annual tax revenue from the entire mobile phone industry, which is about 20billion UK pounds.  In theory the Government could close down the entire mobile phone industry and actually show a profit!

There are ways in which the modulation of the signal can be changed to avoid this, but in the meantime, the compulsory introduction of smart meters can only contribute further to autism on a grand scale. This will be a further burden on the economy and increase the National deficit. This will far outweigh any possible advantages from the use of these meters.

There is also a risk of legal complications for the utility companies. If it can be shown that that the consumer has taken reasonable precautions to minimise their microwave exposure by eliminating WiFi, cordless phones and wireless baby monitors from their house, the utility company could be held legally responsible for any autistic children that they may have.

In the UK, the lifetime cost of caring for an autistic child is in the region of one million pounds. It would be reasonable to claim compensation for this amount. In the United States, it may also be possible to claim punitive damages if it can be shown that the utility company knew of this risk when they installed or refused to remove a smart meter when requested.

Dr Andrew Goldsworthy

Lecturer in Biology (retired)

 Imperial College London


Amaral DG, Schumann CM, Nordahl CW (2008), Neuroanatomy of Autism, Trends in Neurosciences 31: 137-145

Bawin SM, Kaczmarek KL, Adey WR (1975), Effects of modulated VHF fields on the central nervous system. Ann NY Acad Sci 247: 74-81

Beason RC, Semm P (2002), Responses of neurons to an amplitude modulated microwave stimulus. Neuroscience Letters 333: 175-178

Blackman CF (1990), ELF effects on calcium homeostasis. In: Wilson BW, Stevens RG, Anderson LE (eds) Extremely Low Frequency Electromagnetic Fields: the Question of Cancer. Battelle Press, Columbus, Ohio, pp 189-208

Blackman CF, Benane SG, Kinney LS, House DE, Joines WT (1982), Effects of ELF fields on calcium-ion efflux from brain tissue in vitro. Radiation Research 92: 510-520

Diem E, Schwarz C, Adlkofer F, Jahn O, Rudiger H (2005). Non-thermal DNA breakage by mobile-phone radiation (1800 MHz) in human fibroblasts and in transformed GFSH-R17 rat granulosa cells in vitro. Mutation Research 583: 178-183

Goldsworthy A (2010) , Witness Statement,

Hardell L, Carlberg M (2009), Mobile phones, cordless phones and the risk for brain tumours. Int J Oncology 35: 5-17 DOI: 10.3892/ijo_00000307

Hawley T, Gunner M (2000), How early experiences affect brain development.

Hill EL, Frith U (2003), Understanding autism: insights from mind and brain.  Phil Trans R Soc Lond B 358 281-289

Huttenlocher PR, Dabholkar AS (1997) Regional differences in synaptogenesis in human cerebral cortex.  J Comparative Neurology 387 167-178

Krey JF, Dolmetsch RE (2007) Molecular mechanisms of autism: a possible role for Ca2+ signaling. Current Opinion in Neurobiology. 17: 112-119

Volkow ND, Tomasi D, Wang G, Vaska P, Fowler JS, Telang F, Alexoff D, Logan J, Wong C (2011), Effects of Cell Phone Radiofrequency Signal Exposure on Brain Glucose Metabolism. JAMA. 305 (8):808-813. doi: 10.1001/jama.2011.186


Aloe + Bioflavonoid ( Pectin )

Aloe vera in dermatology- a brief review.

Feily ANamazi MR.

Source–Department of Dermatology, Jondishapur University of Medical Sciences, Ahvaz, Iran.

Abstract—Aloe vera Linne or aloe barbadensis Miller is a succulent from the Aloe family (400 different species), a tropical plant which is easily grown in hot and dry climates and widely distributed in Asia, Africa and other tropical areas. The use of aloe vera is being promoted for a large variety of conditions. The aim of this systematic review was to summarize all dermatology-oriented in vitro and in vivo experiments and clinical trials on aloe vera preparations. Extensive literature search were carried out to identify all in vitro and in vivo studies as well as clinical trials on the subject. Data were extracted from these in a predefined standardized manner. Forty studies were located. The results suggest that oral administration of aloe vera in mice is effective on wound healingcan decrease the number and size of papillomas and reduce the incidence of tumors and leishmania parasitemia by >90% in the liver, spleen, and bone marrow. Topical application of aloe vera is not an effective prevention for radiation-induced injuries and has no sunburn or suntan protection. It can be effective for genital herpes, psoriasis, human papilloma virus, seborrheic dermatitis, aphthous stomatitis, xerosis, lichen planus, frostbite, burn, wound healing and inflammation. It can also be used as a biological vehicle andan anti-microbial and antifungal agent and also as a candidate for photodynamic therapy of some kinds of cancer. Even though there are some promising results with the use of aloe vera for diverse dermatologic conditions, clinical effectiveness of oral and topical aloe vera is not sufficiently and meticulously explored as yet.



Parsley has been used for years as a folk remedy for water retention, coughs, allergy, autoimmune, and chronic inflammatory disorders. Although it is one of the most potent disease-fighting plants little is known about how parsley works its magic….that is, until now. Recently, scientists set out to examine just one of the many wonders of parsley, its immune effects, in rigorous scientific detail. They looked at how parsley oil interacts with T-cells and B-cells (both types of white blood cells) and macrophages (cells that engulf and eliminate other cells). Conclusion: parsley oil acted in a similar way to drugs that suppress the immune system, like prednisone, but without the harmful side effects. As if that weren’t enough, other studies have shown that parsley has antitumor, antibacterial, and antioxidant properties.

Karimi MH et al. “Parsley and immunomodulation.” Expert Rev Clin Immunol. 2012 May;8(4):295-7.

It is believed that parsley is one of the world’s seven most potent disease-fighting spices [8]. Although parsley has been used to treat allergy, autoimmune and chronic inflammatory disorders, the mechanism underlying its beneficial effects in these immune-mediated diseases have been rarely investigated. Of the various therapeutically beneficial aspects of parsley, we decided to examine the immunomodulatory effects of this plant.


Parsley– Antiinflammatory

 Throughout history, herbs have been utilized as an important constituent of foods, industry and folk medicine. One of the widely used vegetal species in various nations’ medicine is parsley (Petroselinum crispum), which has remedial effects as a powerful diuretic agent [1,2], an abortifacient [3–5] and an expectorant [6,7]. Parsley is a native herb of the central Mediterranean region (southern Italy, Algeria and Tunisia), which is in the Apiaceae family, and is a species of Petroselinum [8]. It is believed that parsley is one of the world’s seven most potent disease-fighting spices [8]. Although parsley has been used to treat allergy, autoimmune and chronic inflammatory disorders, the mechanism underlying its beneficial effects in these immune-mediated diseases have been rarely investigated. Of the various therapeutically beneficial aspects of parsley, we decided to examine the immunomodulatory effects of this plant. In our study, the effects of parsley essential oil on phytohemagglutinin (PHA)-stimulated splenocytes (T cells) and lipopolysaccharide (LPS)-stimulated B cells, as the main effector cells in adaptive immune system, was examined. In addition, the suppressive activity of different concentrations (0.01–100 μg/ml) of parsley essential oil on macrophages and LPS-stimulated macrophages for evaluation of nitric oxide (NO) was studied [9]. The methyl tetrazolium method was performed to survey the proliferation of mitogen-stimulated splenocytes as well as the viability of pretreated macrophages [9]. NO production of both macrophage groups was determined in the Griess reaction [9]. Parsley essential oil suppressed the proliferation of PHA-stimulated splenocytes at all applied concentrationsSimilarly, it had a suppressive effect on the unstimulated and LPS-stimulated splenocytes, but only at high concentrations (10 and 100 μg/ml).NO production by unstimulated and stimulated macrophages was reduced by parsley essential oil; although, in all concentrations, unstimulated ones produced lower amounts of NO compared to the control group. These results can propose the suppressive effect of parsley essential oil on macrophages, as the major cells involved in the innate immune system [9].–The use of immunosuppressive drugs to control unwanted immune responses such as allergies, autoimmune disease and transplant rejection has grown over the past few years. The disadvantages and side effects of any immunosuppressive treatment are a significant and growing anxiety [10]. Some serious side effects including nephrotoxicity, hepatotoxicity, induction of diabetes, hypertension and neurotoxicity have been stated for various immunosuppressive drugs [10,11]. Thus, healthier and lower risk therapeutics are required. In this regard, more attention has been recently made on natural products. For example, the immunosuppressive activity of various herbal plants and ingredients including Achillea talagonica, [12] Plantago ovata [13], Boerhaavia diffusa [14],Stachys obtsicrena [15], Pollen Typhae [10] and Silymarin [16] has been explored.  Parsley has also been shown to possess other biological activities than these described hereSeveral studies have suggested anticancer potential of parsley. By means of ascorbic acid‑induced lipid peroxidation, the antilipoperoxidant activity of parsley extracts has been shown [10,17,18]. The antioxidant activity of parsley essential oil has been confirmed in other investigations. Wong et al. indicated that the phenolic compounds of parsley were responsible for its antibacterial and antioxidant activity [19]. Zhang and his coworkers demonstrated the antioxidant activity of this herb in terms of b-carotene bleaching capacity and free radical scavenging activity [7]. This concept was then supported by further studies [20]. Parsley possesses several flavonoids such as apiin and luteolin, and its essential oil contains apiol and myristicin. These components are believed to be responsible for the therapeutic effects of parsley [17,21]. Kandaswami et alindicated the direct and indirect effects of flavonoids on tumor cells. Their studies showed that the hydroxylation pattern of the B-ring of the flavons and flavonols, such as luteolin and quercetin, seemed to affect their angionesis and anticancer activity, especially the inhibition of protein kinase activity and antiproliferation [22]. Robak and his coworkers believe that flavonoids are the superoxide anion scavengers of the media and this effect can also lead to their anti-inflammatory effects [23]. Daly et al. observed bioactive phytochemicals, including carotenoids, in parsley[6]. Carotenoids were shown to be associated with a low risk of several human chronic disorders including age-related macular degeneration and certain cancers. Matching the wide use of this vegetal species as a diuretic in folk medicine, natriuretic and hypotensive effects of parsley were demonstrated in studies by Kreydiyyeh and Usta, and de Campos et al. [1,2]. Further studies indicated more biological effects of parsley plants, such as provitamine A activity, and influencing the cell signaling pathways [22,23]. In summary, parsley is a plant with various biological activities. With respect to its immunomodulatory effects, we found that its inhibitory effect on PHA-stimulated splenocytes might be due to the production of cytokines such as IFN-g and IL-2, which are vital for T-cell proliferation or it may influence the signaling pathways. Our results indicated that parsley essential oil can modulate the activity of macrophages without exerting cytotoxic effect. The immunomodulatory effect of parsley essential oil and its modulatory effects on NO production and function of macrophages may identify it as a useful natural candidate to treat some autoimmune and allergic diseases; however, its further application needs more investigation.


1 Kreydiyyeh SI, Usta J. Diuretic effect and mechanism of action of parsley. J. Ethnopharmacol. 79, 353–357 (2002).

2 de Campos KE, Balbi APC, De Freitas Alves MJQ. Diuretic and hypotensive activity of aqueous extract of parsley seeds (Petroselinum sativum Hoffm.) in rats. Braz. J. Pharmacog. 19(1A), 41–45 (2009).

3 Tyler VE. The Honest Herbalist (3rd Edition). Pharmaceutical Products Press, London, UK, 235–236 (1993).

4 Anderson LA, Newall CA, Phillipson JDA. Guide for Health-care Professionals. The Pharmaceutical Press, London, UK, 203–204 (1996).

5 Robbers JE, Tyler VE. Tyler’s Herbs of Choice. The Therapeutic Use of Phytochemicals. Haworth Herbal Press, NY, USA, 92 (1999).

6 Daly T, Jiwan MA, O’Brein M, Aherne SA. Carotenoid content of commonly consumed herbs and assessment of their bioaccessibility using an in vitrodigestion model. Plant. Foods Hum. Nutrit. 65(2), 164–169 (2010).

7 Zhang H, Chen F, Wang X, Yao HY. Evaluation of antioxidant activity of parsley (Petroselinum crispum) essential oil and identification of its antioxidant constituents. Food Res. Int. 39(8), 833–839 (2006).

8 Lopez MG, Sanchez-Mendoza IR, Ochoa-Alejo N. Compartive study of volatile components and fatty acids of plants and in-vitro cultures of parsley (Petroselinum crispum [Mill] nym ex hill). J. Agric. Food Chem. 47, 3292–3296 (1999).

9 Yousofi A, Daneshmandi S, Soleimani N, Bagheri K, Karimi MH. Immunomodulatory effect of Parsley (Petroselinum crispum) essential oil on immune cells: mitogen-activated splenocytes and peritoneal macrophages. Immunopharmacol. Immunotoxicol. 34(2), 303–308 (2012).

10 Vial T, Descotes J. Immunosuppressive drugs and cancer. Toxicology 185(3), 229–240 (2003).

11 Qin F, Sun HX. Immunosuppressive activity of pollen typhae ethanol extract on the immune responses in mice. J. Ethnopharmacol. 102, 424–429 (2005).

12 Rezaeipoor R, Saeidnia S, Kamalinejad M. Immunosuppressive activity of Achillea talagonica on humoral immune responses in experimental animals. J. Ethnopharmacol. 65, 273–276 (1999).

13 Rezaeipoor R, Saeidnia S, Kamalinejad M. The effect of Plantago ovata on humoral immune responses in experimental animals. J. Ethnopharmacol. 72, 283–286 (2000).

14 Mehrotra S, Mishra KP, Maurya R, Srimal RC, Singh VK. Immunomodulation by ethanolic extract of Boerhaavia diffusa roots. Int. Immunopharmacol.2, 987–996 (2002).

15 Amirghofran Z, Bahmani M, Azadmehr A, Javidnia K. Immunomodulatory and apoptotic effects of Stachys obtusicrena on proliferative lymphocytes.Med. Sci. Monit. 13(6), BR145–BR150 (2007).

16 Gharagozloo M, Velardi E, Bruscoli S et al. Silymarin suppress CD4T cell activation and proliferation: effects on NF-kB activity and IL-2 production.Pharmacol. Res. 61(5), 405–409 (2010).

17 Mimica-Dukić N, Popović M. Apiaceae species. A promising sources of pharmacologically active compounds I: Petrosellinum crispumApium greveolensand Pastinaca sativa. In: Recent Progress in Medicinal Plants. Govil JN, Singh VK (Eds). Studium Press LLC, TX, USA (2007).

18 Fejes S, Blázovics A, Lemberkovics E, Petri G, Szöke E, Kéry A. Free radical scavenging and membrane protective effects of methanol extracts fromAnthriscus cerefolium L. (Hoffm.) and Petroselinum crispum (Mill.) Nym. ex A. W. Hill. Phytother. Res. 14(5), 362–365 (2000).

19 Wong PYY, Kitts DD. Studies on the dual antioxidant and anti bacterial properties of parsley (Petroselinum crispum) and cilantro (Coriandrum sativum) extracts. Food Chem. 97, 505–515 (2006).

20 Kolarovic J, Popovic M, Zlinská J, Trivic S, Vojnovic M. Antioxidant activities of celery and parsley juices in rats treated with doxorubicin. Molecules 15, 6193–6204 (2010).

21 Lombaert GA, Siemens KH, Pellaers P, Mankotia M, Ng W. Furanocoumarins in celery and parsnips: method and multiyear Canadian survey. J. AOAC Int. 84, 1135–1143 (2001).

22 Kandaswami C, Lee LT, Lee PP et al. The antitumor activities of flavonoids. In Vivo 19(5), 895–909 (2005).

23 Robak J, Rys Z, Gryglewski J. Flavonoids are scavengers of super oxide anions. Biochem. Pharm. 37, 837–841 (1998)



 [U1]Again this information is somewhat outdated—since  mercury is in most fish consumed today it is arguably that the mercury can further diminish your low iodine levels—so again an alternative maybe eating grass fed beef which will have iodine in them based on the feed and without the mercury or seek other sources of aquatic life that may feed on seaweed to obtain the iodine

 [U2]Radishes can be goiterogenic so again if you do consume them not often and should be utilizing seaweed or watercress with it

 [U3]These are the goiterogenic goods

 [U4]Smart Meter-Cell Phone—HAARP -Microwaves

 [U5]A bad source—you would need to take 650grams -1 lb 7oz-to get the equivalent 1 drop of lugols

 [U6]The key word  is EXCESS!!

 [U7]AGAIN this is only if the pollution is low and fish all has some form of arsenic and mercury so if  you eat fish  make sure that it is marinated properly with anti mercury substances—so that when being consumed the toxins are neutralized

 [U8]Now this is where it gets tricky—it does not say how long it will take—but it gives the feeling or perspective that it will be quick—this IS NOT SO—it will be contingent on health—how much you are lacking—and what other complimentary minerals or aminos you may be  missing as well

 [U9]Other then Watercress the rest of the foods mentioned will be determined in there geographical areas which will determine how much is in the soil And how much is absorbed if any—the iodine that is

 [U10]ACTIVATE the Thyroid

 [U11]Even this is to low way to low-1000mcgs = 1mg the suggested daily dose is 13 mgs to sustain adequate levels—and use more depending on life style and exposure to environments

Posted in Health Politics, Nutrition, Remedies, The Remedy Show notes, Tony Pantellresco's Articles | Leave a comment

Tony Pantalleresco Show Notes for week of 12th of October 2012

How to Self-Test for an Iodine Deficiency

List of factors that impairs thyroid function

 Adrenal Support

Exploring many uses for topical iodine solution

Inactivation of human viruses by povidone-iodine in comparison with other antiseptics

How to Self-Test for an Iodine Deficiency

1. Dip a cotton ball into USP Tincture of Iodine.

2. Paint a 2 inch circle of iodine on your soft skin, like the inner part of your thigh or upper arm.

3. Wait. — If the yellowish stain disappears in less than an 4 hours; it means your body is lacking crucial iodine and has soaked it up. If the stain remains for more than 4-8 hours, you iodine levels are fine.


Why check your iodine levels?—Low iodine levels can zap your energy and make you feel tired, edgy and worn out. Low iodine levels can even prevent you from getting a good night’s sleep. Before you go to your doctor with complaints of tossing and turning all night, aches and pains, and just feeling “blah,” you may want to perform this self-test.—Because the symptoms of an iodine deficiency are classically identical to so many other illnesses (like depression, stress, chronic fatigue, or fibromyalgia,) many doctors either misdiagnose it or miss it completely and tell you there is nothing wrong.


Why are iodine levels so important?–Low levels of iodine mean your thyroid may not be functioning properly. The thyroid needs iodine to function as it helps balance hormones, regulate heartbeats, stabilize cholesterol, maintain weight control, encourage muscle growth, keep menstrual cycles regular, provide energy, and even helps you keep a positive mental attitude.—-Women are naturally prone to iodine deficiencies. That’s because the thyroid gland in women is twice as large as in men — so under normal circumstances, women need more iodine. However, when women are under stress, the need for iodine can double or triple. Yet the foods we eat contain less and less dietary iodine. For example, back in 1940, the typical American diet contained about 800 micrograms of iodine. By 1995, that amount plunged to just 135 micrograms. That’s an 83% decline.—-Two thirds of the body’s iodine is found in the thyroid gland. One of the best ways to boost your iodine levels is to add seaweed -sea vegetables to your diet. Just one teaspoon of sea vegetables a day can help you regain normal iodine levels. Incorporating seafood and fish into your diet can also help[U1] . Other foods that contain iodine are eggs and dairy products, including milk, cheese and yogurt, onions, radishes[U2] , and watercress. Some foods, called goitrogens, should be omitted for awhile as they hinder iodine utilization. These included kale, cabbage, peanuts, soy flour, Brussels sprouts, cauliflower, broccoli, kohlrabi and turnips.—[U3] To reactivate the thyroid gland, tyrosine, iodine, zinc, copper and selenium are needed so make sure that foods containing these nutrients are included in your diet.  However, if you have the immune system deficiency called Hashimoto’s Thyroiditis, you should not supplement your diet with iodine as it may aggravate the condition.


List of factors that impairs thyroid function.

    1. Aging linked to a gradual decline of both thyroid and adrenal hormones.

  2. Alpha Lipoic Acid (does not affect everyone)

  3. Alcohol

  4. Chronic illness

  5. Cigarette smoking

  6. Diet factors. Soy products and cruciferous vegetables ­ dose dependent (avoid large quantities). Studies in animals show soy impairs T4 to T3 conversion.

  7. Drugs (Birth control pills, Lithium, Estrogens, Propranolol, Beta blockers, Dexamethasone, Methimazole and Propylthiouracil)

  8. External radiation[U4] 

  9. Growth hormone deficiency

  10. Heavy metal toxicity including mercury and lead, pesticides, sodium chloride as well as sodium fluoride in city water.

  11. Hemochromatosis

  12. High Stress

  13. Low adrenal states

  14. Malnutrition (mineral deficiencies) consumption of trans fats and hydrogenated fats and a lack of good fats ­ monounsaturated ­ avocados, olive oil, palm, omega 3.

  15. Mineral and vitamin deficiencies (selenium, Vitamin A, B6 and B12)

  16. Postoperative state

  17. Physical trauma.

Besides the above list  research views that in a state of hypothyroidism, hydrocortisone production and metabolism is usually low. Hypothyroidism can be established by chronic low basal body temperature as measured by the Barnes method upon rising. Thus a direct link has been established between subnormal thyroid and inadequate adrenal production of hormones.

Toxemia or the buildup of toxins in the body and an impairment of the detoxification pathways can be a major cause of impaired thyroid function. Link by link, the health of one organ affects the health of another. The liver, the organ through which most detoxification occurs, has the greatest burden of all.

Today, I reasonably estimate that due to toxins in the diet, air pollution and contaminants in the water we drink and electromagnetic pollution, that the combined effect contributes to overworked adrenal and subnormal thyroid activity in at least a third or more of the population in the United States.


Adrenal Support

 Degeneration of the Adrenal Glands may occur as a symptom of Choline deficiency.  [more info]

Vitamin A concentrates in the Adrenal Glands and may improve the function of the Adrenal Glands.  references

Vitamin B5 may activate the Adrenal Glands and may “revive” exhausted Adrenal Glands.

 Vitamin C is essential for the function of the Adrenal Glands – the Adrenal Glands (especially the Adrenal Medulla) contain approximately 30 mg of Vitamin C – the second highest concentration of Vitamin C of any component of the body. 

Vitamin E concentrates in the Adrenal Glands (within the Adrenal Cortex).  The Adrenal Glands contain 132 mcg of Vitamin E per gram). 

Adrenal Extract may improve the function of the Adrenal Glands in persons with impaired Adrenal Gland function.

Tyrosine may relieve excessive stress on the Adrenal Glands

 Pregnenolone production (from Cholesterol) occurs primarily in the Adrenal Glands (but is also produced in smaller quantities in other areas of the body, including the Liver, Brain, Skin and Retina of the Eye).


Could painting a circle of iodine solution on the skin daily help get rid of the candida albicans, parasites, HHV-6, herpes, hepatitis and multiple other bacterial, fungal, parasitic and viral infections including HIV?

Exploring many uses for topical iodine solution

  Iodine for HIV – to directly kill the virus

  Iodine to treat opportunistic infections in AIDS

  Iodine for chronic Candidiasis, EBV and CMV

  Iodine for viral hepatitis A, B, C and D

  Iodine for HHV-6 infection

  Iodine for parasites

  Iodine for herpes

  Iodine for strep and staff infections

  Iodine for sore joints and arthritis

  Iodine for rheumatism

  Iodine for the flu

  Iodine for a sore throat

  Iodine for almost any bacterial, fungal, parasitic or viral infection that exists.

  Iodine to increase the thyroid’s production of thyroxin

  Iodine to help restore normal body temperature

  Iodine to prevent and treat metabolic syndrome (obesity, diabetes and heart disease)

  Iodine is cheap, plentiful and safe to use.

Here is how to do iodine testing and use

1. Dip a cotton ball into USP Tincture of Iodine –Lugols will do as well

2. Paint a 2 inch circle of iodine on your soft skin, like the inner part of your thigh or upper arm.

3. Wait. — If the yellowish stain disappears in less than an hour; it means your body is lacking crucial iodine and has soaked it up. If the stain remains for more than 4-8 hours, your iodine levels are fine.

Why check your iodine levels? —Low iodine levels can zap your energy and make you feel tired, edgy and worn out. Low iodine levels can even prevent you from getting a good night’s sleep. Before you go to your doctor with complaints of tossing and turning all night, aches and pains, and just feeling “blah,” you may want to perform this self-test.

Because the symptoms of an iodine deficiency are classically identical to so many other illnesses (like depression, stress, chronic fatigue, or fibromyalgia,) many doctors either misdiagnose it or miss it completely and tell you there is nothing wrong. —About 60% of all iodine in the human body is stored in the thyroid gland at the base of the neck. A Goiter, an enlargement of the thyroid gland, can occur when iodine deficiency is prolonged over a period of timeIodine is the most important mineral used by the thyroid gland to produce thyroxin, a hormone that regulates the metabolic rate of energy production in the cells. —Iodine deficiency and a resulting insufficient production of thyroxin is linked to a wide range of illness form chronic infection of all types including candidiasis, herpes, bacterial, fungal and viral infections. —Table salt is iodized and is a source of iodine[U5] . However, excess sodium intake from salt consumption is a toxin and impairs the production of energy in the cellsExcess sodium intake is linked to metabolic syndrome including obesity, high blood pressure, heart disease and cancer.  [U6] The best sources of iodine are from sea vegetables and seafood including ocean fish[U7] . Unfortunately, most people do not consume iodine rich foods on a daily basis. Low body temperature and long term chronic infections may be directly linked to a deficiency of iodine and an impaired thyroid function.

How much iodine to consume —-Why are iodine levels so important? Low levels of iodine mean your thyroid isn’t functioning properly. The thyroid helps balance hormones, regulate heartbeats, stabilize cholesterol, maintain weight control, encourage muscle growth, keep menstrual cycles regular, provide energy, and even helps you keep a positive mental attitude. —Women are naturally prone to iodine deficiencies. That’s because the thyroid gland in women is twice as large as in men — so under normal circumstances, women need more iodine. However, when women are under stress, the need for iodine can double or triple. Yet the foods we eat contain less and less dietary iodine. For example, back in 1940, the typical American diet contained about 800 micrograms of iodine. By 1995, that amount plunged to just 135 micrograms. That’s an 83% decline. —-Two thirds of the body’s iodine is found in the thyroid gland. One of the best ways to boost your iodine levels is to add seaweed sea vegetables to your diet. Just one teaspoon of sea vegetables a day can help you regain normal iodine levels[U8] . Incorporating seafood and fish into your diet can also help. Other foods that contain iodine are eggs and dairy products, including milk, cheese and yogurt, onions, and watercressSome[U9]  foods, called goitrogens, should be omitted for awhile as they hinder iodine utilization. These included kale, cabbage, peanuts, radishes, soy flour, Brussels sprouts, cauliflower, broccoli, kohlrabi and turnips. —-To reactivate the thyroid gland, tyrosine, iodine, zinc, copper and selenium are[U10]  needed so make sure that foods containing these nutrients are included in your diet. —Editor’s note: The RDA of iodine is 150 mcg. That is just too little, in my opinion. Ideally, the average daily intake of iodine should be closer to 1000 mcg daily and higher than that for stress conditions that rapidly deplete iodine levels[U11] . Persons with systemic infections will need higher amount of iodine as this trace mineral will be rapidly bound to the infectious agents (bacterial, fungal or viral) in the process of destroying them thus leaving less iodine for the thyroid to pick up. While most people have an under active thyroid, a few people have an overactive thyroid but there is no research linking this to excess iodine intake.


Inactivation of human viruses by povidone-iodine in comparison with other antiseptics.

Kawana R, Kitamura T, Nakagomi O, Matsumoto I, Arita M, Yoshihara N, Yanagi K, Yamada A, Morita O, Yoshida Y, Furuya Y, Chiba S.

Dermatology . 1997;195 Suppl 2:29-35.

Morioka Yuuai General Hospital, Japan.

Inactivation of a range of viruses, such as adeno, mumps, rota-, polio- (types 1 and 3), coxsackie-, rhino-, herpes simplex, rubella, measles, influenza and human immunodeficiency viruses, by povidone-iodine (PVP-I) and other commercially available antiseptics in Japan was studied in accordance with the standardized protocol in vitro. In these experiments, antiseptics such as PVP-I solution, PVP-I gargle, PVP-I cream, chlorhexidine gluconate, alkyldiaminoethyl-glycine hydrochloride, benzalkonium chloride (BAC) and benzethonium chloride (BEC) were used. —-PVP-I was effective against all the virus species testedPVP-I drug products, which were examined in these experiments, inactivated all the viruses within a short period of time. Rubella, measles, mumps viruses and HIV were sensitive to all of the antiseptics, and rotavirus was inactivated by BAC and BEC, while adeno-, polio- and rhinoviruses did not respond to the other antiseptics. PVP-I had a wider virucidal spectrum, covering both enveloped and nonenveloped viruses, than the other commercially available antiseptics.

Posted in Health Politics, Nutrition, Remedies, The Remedy Show notes, Tony Pantellresco's Articles | Leave a comment

Tony Pantalleresco Show Notes – Show Of the Month October 8 2012

Show Of the Month October 8 2012

Black Seed Plus Sarsparilla-Liver Protection

Recipe for making this decoction of Black seed and Smilax ( sarsaparilla )

Symptoms of chemtrail spraying

Your filter specification- Drug Class~~Name- Mood Stabilizer

Your filter specification- Drug Class-Name- Antidepressant


Black Seed Plus Sarsparilla-Liver Protection

Modulation of apoptosis in human hepatocellular carcinoma (HepG2 cells) by a standardized herbal decoction of Nigella sativa seeds, Hemidesmus indicus roots and Smilax glabra rhizomes with anti- hepatocarcinogenic effects.

BMC Complement Altern Med. 2012;12:25

Authors: Samarakoon SR, Thabrew I, Galhena PB, Tennekoon KH

Abstract BACKGROUND: A standardized poly-herbal decoction of Nigella sativa seeds, Hemidesmus indicus roots and Smilax glabra rhizome[U19] s used traditionally in Sri Lanka for cancer therapy has been demonstrated previously, to have anti-hepatocarcinogenic potential. Cytotoxicity, antioxidant activity, anti-inflammatory activity, and up regulation of p53 and p21 activities are considered to be some of the possible mechanisms through which the above decoction may mediate its anti-hepatocarcinogenic action. The main aim of the present study was to determine whether apoptosis is also a major mechanism by which the decoction mediates its anti-hepatocarcinogenic action. METHODS: Evaluation of apoptosis in HepG2 cells was carried out by (a) microscopic observations of cell morphology, (b) DNA fragmentation analysis, (c) activities of caspase 3 and 9, as well as by (d) analysis of the expression of pro-apoptotic (Bax) and anti-apoptotic (Bcl-2) proteins associated with cell death. RESULTS: The results demonstrated that in HepG2 cells, the decoction can induce (a) DNA fragmentation and (b) characteristic morphological changes associated with apoptosis (nuclear condensation, membrane blebbing, nuclear fragmentation and apoptotic bodies). The decoction could also, in a time and dose dependent manner, up regulate the expression of the pro-apoptotic gene Bax and down regulate expression of anti-apoptotic Bcl-2 gene (as evident from RT-PCR analysis, immunohistochemistry and western blotting). Further, the decoction significantly (p < .001) enhanced the activities of caspase-3 and caspase-9 in a time and dose dependent manner.—CONCLUSIONS: Overall findings provide confirmatory evidence to demonstrate that the decoction may mediate its reported anti-hepatocarcinogenic effect, at least in part, through modulation of apoptosis.—PMID: 22458551 [PubMed – indexed for MEDLINE]

Recipe for making this decoction of Black seed and Smilax ( sarsaparilla)—take equal parts of each herb and boil them down to at least half of the volume of water you put in— I.E 2 pint down to 1 pint or less ( 500mls for the metric to 250 mls)—and then use small amounts 1-2 oz increments 4-5 times daily


Symptoms of chemtrail spraying

by OkanaganChemtrails @ 20.08.10 – 19:05:47

We’ve covered this before on this blog but I believe that one way to put an end to this is to educate people how they are being poisoned. Who in their right mind wouldn’t want spraying to cease once they realize their health is being compromised? So here’s a new article and a few links to refresh everyone.

Chemtrails – Health Effects on the General Population

by Claude-Michel Prévost

Over the past ten years, through research and the personal accounts of many individuals, it has become readily apparent that the aluminum and barium salt mixtures, polymer fibers, toxic chemicals and biologicals sprayed in the atmosphere are the irritants that are either directly or indirectly responsible for health problems on the rise in the United States and elsewhere.

These toxic particulates are rapidly absorbed from the respiratory system and / or the gastrointestinal tract and are deposited in the lungs, muscles, and bone.

This illegal aerial spraying is producing atmospheric and ground conditions detrimental to human and animal health but favorable to the growth of harmful molds / fungus.

This overview is a partial list of health problems reported by private citizens to Chemtrail researchers.

1.Nose and lung bleeds (the latter including several reports from nursing homes of elderly dying from lung bleed outs, we believe being directly attributable to atmospheric aerosols); 2.Asthma and allergies;

3.Allergic bronchopulmonary aspergillosis (ABPA) (fungus on the lungs in both infants and adults),

4.Flu, Bronchitis and Pneumonia (in epidemic proportions, with doctors commenting to their patients on the many weeks it sometimes takes to improve and the lack of effective antibiotics to treat, including reports of pets having the flu, whole families being decimated), meningitis (inflammation / infection of the brain);

5.Upper respiratory symptoms (wheezing, dry cough), including Pulmonary Distress Syndrome (PDS) (in newborns, infants and adults alike), Sudden Infant Death (SIDS), and increased nationwide reports of the sudden death of athletes (reported in the news media as having possibly been attributable directly to air particulates / pollution);

6.Deaths from black mold; black or red mold on food crops (farmers reporting pH changes of soil and water), in buildings and ventilation systems (including school buildings);

7.Arthritis-like symptoms and muscular pain (young and old alike, sometimes crippling, and in pets);

8.Gastrointestinal distress (young and old alike, and in pets);

9.Bladder and yeast infections (includes bed wetting, not just in infants but adults);

10.Extreme fatigue (young and old alike);

11.Ringing of the ears, dizziness (increasingly reported immediately preceding or after a storm or weather system);

12.Eye problems – pink eye, blurred and deteriorating vision / nervous tics after exposure to the air outdoors;

13.Dry / cracking skin and lips, rashes, sores and fungal infections, aging of the skin;

14.Mental confusion / slow thinking and / or the feeling of mentally “being in a fog” (young and old alike, increasingly reported after actually being in heavy mists and fog banks);

15.Autoimmune disorders (Lupus, Crohn’s, Addison’s Disease, Rheumatoid Arthritis, etc.)

Note: Some of the above symptoms / illnesses can be related to other physical / environmental factors such as dehydration.)



Aluminum Symptoms

Excessive amounts of aluminum can result in symptoms of poisoning. The symptoms include constipation, colic, loss of appetite, nausea, skin ailments, twitching of leg muscles, excessive perspiration, and loss of energy. People with aluminum poisoning should discontinue the use of aluminum cookware and the drinking of tap water. Small quantities of soluble salts of aluminum present in the blood causes slow form of poisoning characterized by motor paralysis and areas of local numbness, with fatty degeneration of kidney and liver. There are also anatomical changes in the nerve centres and symptoms of gastro intestinal inflammation.

In the last few years there has been much publicity about aluminum, as well as a tentative connection of aluminum to Alzheimer Disease. According to Dr. Terry L. Franks the clinical picture is clear that Alzheimers is concurrently involved with aluminum toxicity and he also believe it is the major contributing factor to Alzheimers. It will progressively worsen in North America in the coming year because of the pervasive use of aluminum. Aluminum has the tendency to freeze up or irritate nerve endings, producing spasm and contracture. When someone is going through aluminum detoxification can actually look like an advanced case of Alzheimers Disease.


Cooking utensils, antacids, baking powders, antiperspirants, some soft water, aluminum foils, concrete and process foods contain aluminum (table salt, cheese slices individually wrapped), bleached flour, fluoridated water increases leaching of aluminum.

How Aluminum Affects Health

Nervous system

In animal studies, aluminum blocks the action potential or electrical discharge of nerve cells, reducing nervous system activity. Aluminum also inhibits important enzymes in the brain (Na-K-ATPase and hexokinase). Aluminum may also inhibit uptake of important chemicals by nerve cells (dopamine, norepinephrine, and 5-hydroxytryptamine).

Behavioural Effects

Dementia resulting from kidney dialysis related to aluminum toxicity causes memory loss, loss of coordination, confusion and disorientation.

Symptoms of Aluminum Toxicity

Early symptoms of aluminum toxicity include: flatulence, headaches, colic, dryness of skin and mucous membranes, tendency for colds, burning pain in the head relieved by food, heartburn and an aversion to meat. Later symptoms include paralytic muscular conditions, loss of memory and mental confusion. Other symtpoms may include:

Alzheimer’s disease, amyotrophic lateral sclerosis, anaemia, haemolysis, leukocytosis, porphyra, colitis, dental cavities, dementia dialactica, hypo-parathyroidism, kidney dysfunction, neuromuscular disorder, osteomalacia, Parkinson’s disease, ulcers.

Digestive system

Aluminum reduces intestinal activity, and by doing so can cause colic.

Treatmnet of Toxicity

Decreasing contact with and use of aluminum-containing substances will reduce intake and allow more aluminum to leave the body. Oral chelating agents will also help clear aluminum more rapidly. Calcium disodium edetate (EDTA) binds and clears aluminum from the body; this substance is fairly nontoxic and used as the agent for “chelation therapy,: an intravenous treatment used to pull metals such as lead from the body, and more recently used in the treatment of atherosclerosis and cardiovascular diseases.

Deferoxamine, an iron chelator, also binds aluminum. In a study with Alzheimer’s patients, nearly 40 percent of the patients showed an improvement in symptoms with deferoxamine treatment. There is some evidence that intravenous chelation with EDTA helps Alzheimer’s patients. More research is needed to evaluate aluminum’s involvement with this disease. Recovery is excellent for removing heavy metals.

Vitamin C

Has been found to bind aluminum. The average dose, if the patient is relatively comfortable, is about six grams a day. Up to twelve grams is not excessive. The average time on an aluminum detoxification is three to four weeks.


The best way to prevent aluminum buildup is to avoid the sources of aluminum. Eliminating foods that have aluminum additives is probably healthier overall. Not using common table salt is a positive health step as well. Some tap waters contain aluminum, this can be checked. Avoiding aluminum cookware and replacing it with stainless steel, ceramic, or glass is a good idea. Blocking skin and sweat pores with aluminum anti-perspirants.



The possibility of barium poisoning is a reality among people working in and living near heavy industrial sites such as chemical plants, factories that produce rubber products and other such places. That is because barium is one of the components used in manufacturing the products created in these plants.

However, because many of these products end up in ordinary households, it is also possible for a person who does not work in or live near an industrial plant to experience barium poisoning. Rat poison, for instance, contains barium compounds. Some fluorescent bulbs have coatings made from barium oxide. Fish caught in waters near industrial sites may have absorbed barium from the water.

Given the considerable probability of a person becoming afflicted with barium poisoning, how would you know for sure if you or someone living with you ingested barium at toxic levels? If you find yourself or someone living in your household with symptoms of barium poisoning, then you should act immediately.

What Is Barium?

In order to understand how serious barium poisoning is, we need first to understand what barium is in the first place. Barium is a heavy metal that naturally occurs in the environment. It is silvery white in appearance.

Barium is valuable in many industries that make use of heavy metal because it can remove traces of oxygen in some chemical compounds. It also increases the luster of glass. However, barium is explosive and can react violently when mixed with water. Also, it cannot be digested by the body; barium can be poisonous if the amount the body contains exceeds tolerable levels.

Symptoms of Barium Poisoning to Look For

When barium accumulates in the body, it usually affects the functions of the nervous system. Barium poisoning displays symptoms that are similar to flu, which is why it is not strange to find the condition misdiagnosed as flu. Common symptoms of barium poisoning include:

1. Muscle weakness and tremors 2. Difficulty in breathing 3. Stomach irritations accompanied by diarrhea 4. Anxiety 5. Cardiac irregularities such as abnormally high blood pressure and rapid heartbeat 6. Paralysis

What to Do in Case of Barium Poisoning

In case someone in your household has just ingested something that contains barium, the first thing you need to do is to induce him or her to vomit. This will get some of the barium out of the victim’s system. You can also mix a tablespoon of Epsom salts (sodium sulfate or magnesium sulfate) with a glass of water and get the victim to drink it. Afterwards, you should bring the victim to the emergency room of the nearest hospital to make sure that he or she does not succumb to barium poisoning.

You can also prevent barium poisoning from happening in your home. For one, you should keep your rat poison or any other chemical substances in your house that contains barium out of reach of children. Make sure that you have labeled their containers properly.

You should also avoid eating fish that was caught near industrial sites and ascertain that the fish and seafood you eat does not contain barium or any other heavy metal. This will prevent you from accumulating barium in your system and suffer from barium poisoning later on.





[U1]East indian Sarsparilla-chinese Sarsparilla and Black seed-decoction would be a tea boiled down to a complete extract

[U2]The less healthy oils would be soy and vegetable and canola oils or anything hydrogenated—these are toxic and become more so when heated and literally cause Genetic damage to the body

[U3]This is outdated there is no such thing as good or bad cholesterol in fact this study shows that using these


Posted in Health Politics, Nutrition, Remedies, The Remedy Show notes, Tony Pantellresco's Articles | Leave a comment

Tony Pantalleresco Show Notes – Show Of the Month October 5 2012

Diet promotes sleep duration and quality

Short Sleep Duration and Weight Gain– A Systematic Review

Cell tower regulations frustrate homeowners Towers under 15 metres tall avoid municipal scrutiny

Red wine and components flavonoids inhibit UGT2B17 in vitro

Rhodiola Protects HPG Axis during Exercise or Physical Work


Diet promotes sleep duration and quality.

Nutr Res. 2012 May;32(5):309-19–Authors: Peuhkuri K, Sihvola N, Korpela R

Abstract–Sleep, much like eating, is an essential part of life. The mechanisms of sleep are only partially clear and are the subject of intense research. There is increasing evidence showing that sleep has an influence on dietary choices. Both cross-sectional and epidemiologic studies have demonstrated that those who sleep less are more likely to consume energy-rich foods (such as fats or refined carbohydrates), to consume fewer portions of vegetables, and to have more irregular meal patterns. In this narrative review, we pose the opposite question: can ingested food affect sleep? The purpose of this review is to discuss the evidence linking diet and sleep and to determine whether what we eat and what kind of nutrients we obtain from the food consumed before bedtime matter. In addition, scientific evidence behind traditional sleep-promoting foods such as milk and some herbal products is briefly described. These are reviewed using data from clinical trials, mostly in healthy subjects. In addition, we discuss the possible mechanisms behind these observations. Lastly, we summarize our findings that emerging evidence confirms a link between diet and sleep. Overall, foods impacting the availability of tryptophan, as well as the synthesis of serotonin and melatonin, may be the most helpful in promoting sleep. Although there are clear physiological connections behind these effects, the clinical relevance needs to be studied further.—PMID: 22652369 [PubMed – indexed for MEDLINE]


Short Sleep Duration and Weight Gain– A Systematic Review

Sanjay R. Patel1 and Frank B. Hu2,3,4

  1. 1Division of Pulmonary, Critical Care and Sleep Medicine, Department of Medicine, University Hospitals Case Medical Center and Case Western Reserve University, Cleveland, Ohio, USA
  2. 2Channing Laboratory, Department of Medicine, Brigham and Women’s Hospital, and Harvard Medical School, Boston, Massachusetts, USA
  3. 3Department of Nutrition, Harvard School of Public Health, Boston, Massachusetts, USA
  4. 4Department of Epidemiology, Harvard School of Public Health, Boston, Massachusetts, USA

Correspondence: Sanjay R. Patel (

Received 18 April 2007; Accepted 19 July 2007; Published online 17 January 2008.

The recent obesity epidemic has been accompanied by a parallel growth in chronic sleep deprivation. Physiologic studies suggest sleep deprivation may influence weight through effects on appetite, physical activity, and/or thermoregulation. This work reviews the literature regarding short sleep duration as an independent risk factor for obesity and weight gain.

Methods and Procedures—A literature search was conducted for all articles published between 1966 and January 2007 using the search “sleep” and (“duration” or “hour” or “hours”) and (“obesity” or “weight”) in the MEDLINE database. Additional references were identified by reviewing bibliographies and contacting experts in the field. Studies reporting the association between sleep duration and at least one measure of weight were included.

Results—Thirty-six publications (31 cross-sectional, 5 prospective, and 0 experimental) were identified. Findings in both cross-sectional and cohort studies of children suggested short sleep duration is strongly and consistently associated with concurrent and future obesity. Results from adult cross-sectional analyses were more mixed with 17 of 23 studies supporting an independent association between short sleep duration and increased weight. In contrast, all three longitudinal studies in adults found a positive association between short sleep duration and future weight. This relationship appeared to wane with age.

Discussion–Short sleep duration appears independently associated with weight gain, particularly in younger age groups. However, major study design limitations preclude definitive conclusions. Further research with objective measures of sleep duration, repeated assessments of both sleep and weight, and experimental study designs that manipulate sleep are needed to better define the causal relationship of sleep deprivation on obesity.

Introduction—Over the past several decades, the prevalence of obesity has grown to epidemic proportions. Concurrent with this rise in weight there has been a similar epidemic of chronic sleep deprivation. According to annual surveys done by the National Sleep Foundation, by 1998 only 35% of American adults were obtaining 8 h of sleep and that number had fallen to 26% by 2005 (ref. 1[U12] ).—–Evidence has grown over the past decade supporting a role for short sleep duration as a novel risk factor for weight gain and obesity. A number of causal pathways linking reduced sleep with obesity have been posited based on experimental studies of sleep deprivation. Chronic partial sleep deprivation causes feelings of fatigue which may lead to reduced physical activity (2,3). Sleep deprivation may also have neurohormonal effects that increase caloric intake (4). Because of the rapidly accelerating prevalence of sleep deprivation, any causal association between short sleep durations and obesity would have substantial importance from a public health standpoint. We performed a systematic review of the literature to assess the present evidence suggesting that sleep deprivation may represent a novel risk factor for weight gain and obesity.

Methods and Procedures—-Relevant original articles were identified by searching the MEDLINE database (National Library of Medicine, Bethesda, MD) of articles published between 1966 and August 2006 examining the relationship between sleep duration and weight gain, obesity, or both. The primary search was performed using the keywords “sleep” and (“duration” or “hour” or “hours”) and (“obesity” or “weight”). A subsequent search was also performed using medical subheading terms. The searches were repeated in January 2007 to identify any new publications. Bibliographies of retrieved articles were reviewed, and experts in the field were contacted to further identify relevant works. Articles were restricted to studies conducted in humans presenting original research. Where data from the same cohort were presented in more than one article, only the report that most directly analyzed the sleep–weight association was included. All abstracts obtained from this search were screened. Relevant articles were obtained and evaluated for presentation of data regarding the association between sleep duration and at least one measure of weight (e.g., BMI, BMI z-score, and weight) either cross-sectionally or longitudinally. A meta-analysis was attempted, but the degree of heterogeneity among study designs, particularly with respect to the measure of association and the definition of short sleep duration, was prohibitive, and therefore a more qualitative assessment is presented. Greater weight is given to large studies, prospective cohort studies, and studies which objectively assessed sleep durations. Because of differences in the sleep requirements of children and adults, these groups are considered separately. Where results were presented graphically, authors were contacted to obtain the numeric data (3,5,6).

Results—The keyword search initially identified a total of 1,013 citations. After screening through abstracts for relevance, 36 articles of potential relevance were identified. The medical subheading search identified an additional five articles. Fifteen articles were excluded because, though both sleep duration and weight data were collected, the association between these two factors was not assessed. Another five articles were excluded for presentation of data overlapping with another report, leaving 21 articles. Ten investigations were added after the original extraction from review of references and expert contact. The updated search in January 2007 identified 44 additional citations, of which 5 were relevant for this synthesis. Thus 36 studies were included in this analysis. Of these, 31 are cross-sectional studies, 2 are prospective cohort studies, and 3 report both cross-sectional and prospective findings. No experimental studies with weight as an outcome were identified. There are 13 studies examining the association between sleep duration and weight in pediatric populations and 23 studies of adults.

Cross-sectional studies in children—Eleven studies were identified which assessed the cross-sectional association between sleep duration and weight in children (Table 1). All 11 works reported a positive association between short sleep duration and increased obesity. For the most part, obesity was defined by age-adjusted thresholds of BMI, which was directly measured, while sleep duration was typically obtained from questionnaires completed by parents. Because sleep requirements change through childhood, definitions of short sleep duration varied greatly based on the age of the cohort being studied.–The largest pediatric cohort to date is a Japanese birth cohort of 8,274 children assessed between the ages of 6 and 7 (ref. 7). Compared to children with a sleep duration of 10 h, the odds ratios (ORs) for obesity were 1.49, 1.89, and 2.89 for sleep durations of 9–10, 8–9, or <8 h, respectively. A study of 4,511 Portuguese school children aged 7–9 reported similar findings (8). Compared to a sleep duration of 11 h, the ORs for obesity were 2.27 and 2.56 for sleep durations of 9–10 and 8 h, respectively.—-Two studies have analyzed data from children undergoing health screens at school enrollment. A study of 6,645 German children aged 5–6 years found the ORs for obesity were 1.18 and 2.22 for sleep durations of 10.5–11.0 and <10.5 h, respectively, compared to 11.5 h (9). A similar study of 1,031 French 5-year-olds found the OR for obesity was 1.4 for a sleep duration <11 h (10).—Three smaller studies have examined a broader range of grade school children. A study of 422 Canadian children ages 5–10 found that compared to a sleep duration of 12 h, the ORs for obesity were 1.42 and 3.45 for sleep durations of 10.5–11.5 and 10 h (11). Two small case–control studies of children aged 6–10 years, one from Brazil and one from Tunisia, reported similar findings. Giugliano and Carneiro reported obese children had 31 min shorter sleep duration than normal weight children but no significant difference was found between overweight and normal weight children (12). Ben Slama et al. found 58% of obese children had a sleep duration <8 h compared to only 11% of nonobese children (13).

Four studies have examined the relationship between sleep duration and weight in adolescents. Two of these studies, though small, were notable for using objective measures of sleep habits. Measuring sleep duration with wrist actigraphy over a 24-h period in 383 children aged 11–16, Gupta et al. reported one of the strongest associations between short sleep duration and obesity, with the odds of obesity increasing five-fold for every hour reduction in sleep duration (14). Benefice et al., using an accelerometer worn near the hip to assess sleep over 3–4 days in 40 Senegalese girls aged 13–14 years, observed that sleep duration was reduced by 6.85 min for every 1 kg/m2 increase in BMI (15). This work was notable for demonstrating a sleep–weight relationship in a nonobese population—mean BMI was only 16.9 kg/m2. The other adolescent reports included one of 4,486 American teens (mean age 16.6 years), which found short self-reported sleep duration predicted both higher BMI z-score and overweight among boys. However, no relationship was found in girls (16). A study of 656 Taiwanese teenagers (mean age 15.0 years) found that the frequency of obtaining a sleep duration of at least 6–8 h was inversely correlated with obesity risk (17).

The consistent findings from studies spanning five continents suggest that the reported associations are independent of ethnicity, though no formal assessment of effect modification by race has been reported. Several studies suggest boys may be more susceptible to sleep loss than girls. Sekine et al. found the OR for obesity associated with a sleep duration <8 h compared to >10 h was 5.5 in boys and 2.1 in girls (7). Similarly, Chaput et al. found that the OR for obesity associated with a sleep duration of 10 h as opposed to 12 h was 5.7 in boys and 3.2 in girls (11). Knutson found the risk of being overweight increased 10% for each hour reduction in sleep duration among boys, while no significant effect was found among girls (16). A few studies have attempted to identify the causal pathway linking sleep duration to obesity. Von Kries et al. found no relationship between sleep habits and caloric intake obtained from a food frequency questionnaire (9). Gupta et al., using actigraphy, and Benefice et al., using accelerometry to estimate activity levels, each found no relationship between sleep duration and physical activity (14,15).

Cross-sectional Studies in adults—Nineteen studies have focused on the cross-sectional relationship between sleep duration and weight in adults. The findings have been less consistent than the pediatric literature. Eleven studies reported a clear association between short sleep duration and increased weight, and two studies reported mixed findings with an association found in one gender but not in the other. Five studies reported no association between short sleep duration and increased weight, while one found short sleep duration was associated with reduced weight. In addition, six studies have found evidence that long sleep durations are also associated with increased weight resulting in a U-shaped curve between sleep duration and weight. In general, obesity has been defined as a BMI 30 kg/m2 based on either measured or self-reported height and weight. Habitual sleep duration has been typically obtained through questionnaire.—-The largest studies reporting on the association between sleep duration and weight were designed as prospective cohort studies to examine the effects of a wide range of behaviors on health outcomes and were not specifically designed to study sleep duration (5,18,19,20). Furthermore, data on the cross-sectional association between sleep duration and weight in these cohorts were presented as part of analyses designed to assess sleep duration as a predictor of mortality and so focused on the potential of weight to confound the sleep–mortality association. As a result, only the marginal associations between sleep duration and BMI were computed. The largest of these studies was a survey by the American Cancer Society of over 1.1 million individuals (5). This study found a U-shaped association between sleep duration and BMI among women with the minimum at 7 h and a monotonic trend in men such that longer sleep durations were associated with a lower BMI. Comparing a sleep duration of 4–7 h, women had a 1.39 kg/m2 greater BMI and men had a 0.57 kg/m2 greater BMI. The next largest study was a Japanese cohort of over 100,000 individuals (19). This is the only study to find short sleep durations associated with reduced weight. Mean BMI in those with sleep durations 4, 5, 6, and 7 h were 22.2, 22.6, 22.9 and 22.7 kg/m2 for men and 22.6, 22.9, 22.9, and 22.9 kg/m2 for women. A second Japanese cohort of over 10,000 individuals found no association between sleep duration and weight (20). On the other hand, a Scottish study of 6,797 individuals found mean BMI was 0.3 kg/m2 greater among men with a sleep duration <7 h compared to 7–8 h (18). Two other studies considered weight as a secondary outcome. The Sleep Heart Health Study, in studying the association of sleep duration with hypertension, found a U-shaped association between sleep duration and weight with BMI 0.7 and 0.4 kg/m2 greater in those with sleep durations <6 and 6–7 h compared to 7–8 h (21). A Swedish study of sleep duration and diabetes found sleep duration was inversely correlated with both BMI (r = -0.06) and waist-to-hip ratio (r = -0.08) (ref. 22).

Two studies using population-based sampling techniques have directly assessed the relationship between short sleep duration and obesity in middle-aged populations. The larger studied 3,158 adults and found an inverse association between sleep duration and obesity with a minimum risk associated with a sleep duration of 8–9 h (23). Compared to this group, the ORs for obesity were 1.85, 1.49, 1.24, and 1.09 for sleep durations 5, 5–6, 6–7, and 7–8 h. A second study of 1,772 Spanish subjects found a similar association with the odds of obesity 39% greater in those with a sleep duration of 6 h compared to a sleep duration of 7 h.

Several studies have examined the sleep–weight association in working populations. A survey of 4,878 Brazilian truck drivers found a sleep duration <8 h per day was associated with a 24% greater odds of obesity (24). Similarly, a survey of 4,793 Hong Kong union members found an inverse correlation between sleep duration and BMI (r = -0.037, P = 0.02) (ref. 25). This relationship was almost exclusively observed in men. In contrast, a French study of 3,127 workers found that while no association between sleep duration and weight was found in men, among women, those with a sleep duration of 6 h had a 0.63 kg/m2 greater mean BMI than those with longer sleep durations (26). In a study of 1,024 government workers in Wisconsin, a U-shaped association was found using sleep duration based on sleep diaries (27). In multivariate modeling, the minimum BMI corresponded to a sleep duration of 7.7 h. A cross-sectional study of 990 employed adults in Iowa found that BMI was 0.42 kg/m2 greater for each hour reduction in sleep duration (28).

Analysis of a Canadian family-based cohort supports the presence of a U-shaped relationship between sleep duration and obesity (29). The ORs for obesity were 1.63 and 1.51 in women with sleep durations of 5–6 and 9–10 h compared to 7–8 h. The corresponding values in men were 1.72 and 1.18. Similar associations were found between sleep duration and waist-to-hip ratio, body fat mass, and skinfold thicknesses.

Two reports have specifically examined the association between sleep duration and weight in geriatric cohorts. Both were designed to define normative sleep habits in the elderly and considered weight as a predictor of sleep duration. The first study recruited 8,091 individuals over the age of 55 from seven European nations (30). Obesity did not predict being in the lowest 5th percentile of sleep durations. A study of 1,026 French subjects over 60 found those with a BMI >27 kg/m2 were 3.6 times more likely to report nocturnal sleep duration in the lowest 5th percentile than those with BMI of 20–25 kg/m2 (31). However, the obese were also more likely to report daytime naps so that no association existed between total sleep duration and obesity.–Only one study of adults has examined the sleep–weight relationship using an objective measure of sleep duration. Lauderdale et al. investigated predictors of sleep duration in 669 individuals and used 72-h actigraphy to assess average sleep duration (32). In multivariate analysis, the study found a weak inverse correlation between sleep duration and BMI that was not statistically significant.—Two studies have examined the association between sleep habits and weight in clinic populations. Among 924 Americans attending a primary care clinic, sleep duration was longest in those with BMI <25 kg/m2 (33). In a study of 453 Japanese clinic patients, the odds of obesity was nearly double in those with a sleep duration <6 h (34).—Overall, the cross-sectional data in adults suggest short sleepers are heavier though the findings are much less consistent than the pediatric data. Several reports have noted a U-shaped association between sleep duration and weight in adults with the lowest BMI associated with a sleep duration of 7–8 h (5,21,27,29). If this relationship is truly U-shaped, studies that force a linear relationship in modeling the sleep–weight association would underestimate the true effect of short sleep duration and might explain the negative findings in some studies. Ethnic differences in susceptibility to sleep deprivation may also explain the disparate findings, as two of the three Japanese studies were negative. Although no study has directly examined differential susceptibility by ethnicity, several studies have noted that both obesity and sleep deprivation are more common among African Americans than whites (23,32). Findings on differences in gender susceptibility have been mixed. While several studies suggested a greater vulnerability in women (5,26,29,33), at least two reports found an association between short sleep duration and obesity existed only in men (18,25).—In terms of understanding the mechanism of any sleep–weight association, four of the studies finding an association between short sleep duration and obesity found this association could not be explained by differences in physical activity (18,26,29,35). In addition, one of the negative studies also found no relationship between sleep duration and physical activity (32). None of the studies assessed caloric intake. However, two studies examined biomarkers that may be relevant to appetite. Short sleep durations were associated with suppressed leptin levels in both the Quebec Family Study and the Wisconsin Sleep Cohort Study after adjusting for obesity (27,29). Short sleep durations were also associated with elevated ghrelin levels in the Wisconsin cohort (27).


Cell tower regulations frustrate homeowners Towers under 15 metres tall avoid municipal scrutiny

Cellular towers sprouting around Canadian churches

Cell tower in church parking lot draws ire of neighbours

Health Canada: Safety of cellphones and cellphone towers

Do you live near an unexpected cellphone tower?

Suburban cell tower woes4:30

Homeowners across Canada are discovering cellphone towers popping up in residential neighbourhoods that slip just under height regulations that would require the company to notify those living nearby. Oakville[U13] , Ont., resident Lisa Guglietti was in the midst of building her dream home when the mother of three noticed eight cellular network antennas strapped to the chimney of a Bell Canada building, a short distance from her son’s bedroom.  “We were surprised that we weren’t notified,” she said. “We asked some of the neighbours. None of the neighbours had any clue that these cellular antenna had been put up.” Under federal regulations, cellphone companies must notify the municipality for towers at least 15 metres high, but many new installations are coming up short of the limit, at just 14.9 metres. Homeowners say the rule undermines their ability to weigh in on installations in the community. Though the antennas are an eyesore, Guglietti’s primary concern is possible health effects. Experts disagree on the impact caused by cell towers. The International Agency for Research on Cancer classifies radiofrequency electromagnetic fields, which are emitted by wireless phones and cell towers, as a possible human carcinogen. Health Canada states that radiofrequency fields given off by cellphone towers are safe as long as the facility adheres to federal regulatory requirements limiting human exposure. In an email to CBC News, a Bell spokesperson wrote that all its sites, including the Oakville, Ont., one near Guglietti’s house, “meet or exceed all federal safety and other operating requirements.”

City councillor struggles with issue—In June, construction began on a 14.9-metre cellphone tower in a Barrie, Ont. neighbourhood that triggered a backlash over potential health concerns for those living across the street and students walking to nearby schools. “Telecommunications companies are able to come in and put these things basically wherever they want: as close to any residents, as close to any schools, and as close to any community centre they want,” Barrie, Ont. city councillor John Brassard told CBC News. “Why not make it 14.99 metres?” he asked. Since the incident, the Barrie city councillor has begun working to change federal regulations to give Canadians a voice over the placement of cell towers in their neighbourhoods. “Authority and a large part of that decision making should be made by the municipality and in consultation with Industry Canada. Not just Industry Canada alone.”

Government, company response—CBC News requested government data on the number of towers under 15 metres erected across Canada, but Industry Canada said the department doesn’t keep a database of that information[U14] . In the last year, Ottawa has collected about $582 million in revenue from telecommunications companies rolling out their networks of cell towers[U15] . Industry Canada told CBC News that companies are required to consult with the municipality and public before installing antenna towers, unless the towers fall within a certain height.  “Certain installations, including towers less than 15 metres, generally have minimal local impact and so may be excluded from municipal consultation,” an Industry Canada spokesperson said in a written statement to CBC News.  After discovering the cell antennas on the large brick building next door to her new house, Guglietti contacted the federal agency.  An Industry Canada official responded in an email to Guglietti on June 8, 2012 that “given that the installation at the Bell central office building on Balsam Street complies with all procedural and technical requirements, Industry Canada is not in a position to order Bell to relocate the facility.” Cell tower antennas were strapped to a chimney that was 13 metres away from the bedroom of Lisa Guglietti’s son[U16] . (Angela Gilbert/CBC) Guglietti also contacted Bell Canada, which owns the building next door, and says she was initially told it would try to find an alternative location. However, the eight cell antennas remain attached to the chimney next door.

‘Don’t want to be a guinea pig’ —The scientific uncertainty over the health impact of cellphone towers doesn’t sit well with Guglietti.  “I’m supposed to be OK with that?” asked Guglietti. “I’m supposed to have my son exposed to these frequencies day in and day out and I have to wait. Maybe in 10 years from now I’m going to find out, ‘Oh yeah there is, there can be health hazards in living so close to a cell tower.’ ” “I don’t want to be a guinea pig,” said Guglietti.  A Bell spokesperson said in an email to CBC News that cellphone towers are being installed to meet customer demand[U17] .  Guglietti said she’s certain other homeowners are dealing with similar concerns.   If you have any information on this story, or other cellphone tower stories, please contact us at“I need to protect myself and I need to protect my family. I’m a mother and I’m sure anyone would do the same thing in our situation.” If you have any information on this story, or other cellphone tower stories, please contact us at


Red wine and components flavonoids inhibit UGT2B17 in vitro.

Jenkinson C, Petroczi A, Naughton DP.


BACKGROUND: -The metabolism and excretion of the anabolic steroid testosterone occurs by glucuronidation to the conjugate testosterone glucuronide which is then excreted in urine. Alterations in UGT glucuronidation enzyme activity could alter the rate of testosterone excretion and thus its bioavailability. The aim of this study is to investigate if red wine, a common dietary substance, has an inhibitory effect on UGT2B17.

METHODS-Testosterone glucuronidation was assayed using human UGT2B17 supersomes with quantification of unglucuronidated testosterone over time using HPLC with DAD detection. The selected red wine was analysed using HPLC and the inhibitory effects of the wine and phenolic components were tested independently in a screening assay. Further analyses were conducted for the strongest inhibitors at physiologically relevant concentrations. Control experiments were conducted to determine the effects of the ethanol on UGT2B17.

RESULTS-Over the concentration range of 2 to 8% the red wine sample inhibited the glucuronidation of testosterone by up to 70% over 2 hours[U18] . The ethanol content had no significant effect. Three red wine phenolics, identified by HLPC analyses, also inhibited the enzyme by varying amounts in the order of quercetin (72%), caffeic acid (22%) and gallic acid (9%); using a ratio of phenolic: testosterone of 1:2.5. In contrast p-coumaric acid and chlorogenic acid had no effect on the UGT2B17. The most active phenolic was selected for a detailed study at physiologically relevant concentrations, and quercetin maintained inhibitory activity of 20% at 2 M despite a ten-fold excess of testosterone.

CONCLUSION-This study reports that in an in vitro supersome-based assay, the key steroid-metabolising enzyme UGT2B17 is inhibited by a number of phenolic dietary substances and therefore may reduce the rate of testosterone glucuronidation in vivo. These results highlight the potential interactions of a number of common dietary compounds on testosterone metabolism. Considering the variety of foodstuffs that contain flavonoids, it is feasible that diet can elevate levels of circulating testosterone through reduction in urinary excretion. These results warrant further investigation and extension to a human trial to delineate the health implications.


Rhodiola Protects HPG Axis during Exercise or Physical Work

[Salidroside protects the hypothalamic-pituitary-gonad axis of male rats undergoing negative psychological stress in experimental navigation and intensive exercise].

[Article in Chinese]

Wang Q, Wang J, Sun LJ, Hu LP, Li J, Shao JQ, Lu B, Wang YT, Wu B, Wang GH.

Source-Department of Endocrinology, Nanjing University School of Medicine/Nanjing General Hospital of Nanjing Military Region, Nanjing, 210002 Jiangsu, China.

OBJECTIVE-To study the effects of salidroside on the function and ultramicro-pathological change of the hypothalamic-pituitary-gonadal (HPG) axis of male rats in experimental navigation and intensive exercise.

METHODS-Six-week SD rats were randomized into 3 groups: non-stress control (NC, n = 10), training control (TC, n = 12) and salidroside treatment (ST, n = 12) group. Blood samples were collected from the NC rats that did not receive any stimulus after a 7-day intragastric administration of saline. The TC rats underwent a 10-day running training with increasing load on the treadmill followed by a 7-day intragastric administration of saline. The ST rats were subjected to the same process of running training as the TC group and received intragastric administration of salidroside. Then blood samples were immediately obtained and the levels of testosterone (T), corticosterone (CORT), adrenocorticotropic hormone (ACTH), luteinizing hormone (LH) and gonadotropin-releasing hormone (GnRH) measured by radioimmunoassay. The testis histopathology was observed by HE staining, and the ultrastructural changes of the pituitaries and testes investigated by electron microscopy.

RESULTS-The serum T level was significantly lower in the TC than in the NC group, but showed no significant difference between the ST and NC groups. HE staining revealed no significant difference in testis histopathology among the 3 groups. Ultramicro-pathology showed that the secretory granules of the pituitary cells were significantly reduced in the TC rats compared with the NC ones; the number of the granules significantly increased in the ST group compared with the TC rats; and mitochondrial swelling, increase of electron density and decrease/disappearance of mitochondrial cristae were observed in the Leydig cells of the TC rats. But no significant differences were found in the testicular cells between the ST and NC groups.

CONCLUSION-Negative psychological stress and intensive exercise can significantly suppress the function of the HPG axis in rats. Salidroside therapy has protective effect on the HPG axis.

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